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Genome Research|December 13, 2016
APOBEC3A/B-induced mutagenesis is responsible for 20% of heritable mutations in the TpCpW contextVladimir B Seplyarskiy, Maria A Andrianova, Georgii A BazykinGenome Research|May 18, 2017
Human mismatch repair system balances mutation rates between strands by removing more mismatches from the lagging strandMaria A Andrianova, Georgii A Bazykin, Sergey I Nikolaev, et al.Genome Biology and Evolution|February 10, 2018
Are Nonsense Alleles of Drosophila melanogaster Genes under Any Selection?Nadezhda A Potapova, Maria A Andrianova, Georgii A Bazykin, et al.Biorxiv : the Preprint Server for Biology|November 19, 2025
Segregating DNA lesions point to high selective advantage of tumor initiating cellsVladimir Seplyarskiy, Maha Shady, Maria A Andrianova, et al.Nature Genetics|December 5, 2018
Error-prone bypass of DNA lesions during lagging-strand replication is a common source of germline and cancer mutationsVladimir B Seplyarskiy, Evgeny E Akkuratov, Natalia Akkuratova, et al.European Journal of Human Genetics : EJHG|April 24, 2024
Discovery of recessive effect of human polymerase δ proofreading deficiency through mutational analysis of POLD1-mutated normal and cancer cellsMaria A Andrianova, Vladimir B Seplyarskiy, Mariona Terradas, et al.The Journal of Pathology|August 15, 2017
Germline PMS2 and somatic POLE exonuclease mutations cause hypermutability of the leading DNA strand in biallelic mismatch repair deficiency syndrome brain tumoursMaria A Andrianova, Ghati Kasturirangan Chetan, Madathan Kandi Sibin, et al.Nature Genetics|September 12, 2024
Five latent factors underlie response to immunotherapyJoseph Usset, Axel Rosendahl Huber, Maria A Andrianova, et al.Nature Genetics|March 8, 2016
Genomic analysis identifies new drivers and progression pathways in skin basal cell carcinomaXimena Bonilla, Laurent Parmentier, Bryan King, et al.Pageof 1