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Maria G Tsokos

Showing results (31-40 of 51) with videos related to

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Nature Communications|January 29, 2024
CaMK4 controls follicular helper T cell expansion and function during normal and autoimmune T-dependent B cell responsesMarc Scherlinger, Hao Li, Wenliang Pan, et al.
Shock (Augusta, Ga.)|April 19, 2019
Complement Deposition on the Surface of RBC After Trauma Serves a Biomarker of Moderate Trauma Severity: A Prospective StudyAbhigyan Satyam, Kostas Andreo, Peter H Lapchak, et al.
Clinical Immunology (Orlando, Fla.)|July 12, 2021
Complement activation and increased expression of Syk, mucin-1 and CaMK4 in kidneys of patients with COVID-19Simin Jamaly, Maria G Tsokos, Rhea Bhargava, et al.
Journal of Immunology (Baltimore, Md. : 1950)|September 8, 2017
IL-17A Produced by Innate Lymphoid Cells Is Essential for Intestinal Ischemia-Reperfusion InjuryMayya Geha, Maria G Tsokos, Robin E Bosse, et al.
The Journal of Clinical Investigation|May 6, 2021
IL-23 reshapes kidney resident cell metabolism and promotes local kidney inflammationHao Li, Maria G Tsokos, Rhea Bhargava, et al.
JCI Insight|March 30, 2021
Aberrantly glycosylated IgG elicits pathogenic signaling in podocytes and signifies lupus nephritisRhea Bhargava, Sylvain Lehoux, Kayaho Maeda, et al.
Science Translational Medicine|February 19, 2025
Gluconolactone restores immune regulation and alleviates skin inflammation in lupus-prone mice and in patients with cutaneous lupusWei Li, Antonios G A Kolios, Wenliang Pan, et al.
Science Advances|June 15, 2022
CD38 reduces mitochondrial fitness and cytotoxic T cell response against viral infection in lupus patients by suppressing mitophagyPing-Min Chen, Eri Katsuyama, Abhigyan Satyam, et al.
Journal of Immunology (Baltimore, Md. : 1950)|December 4, 2016
Intracellular Activation of Complement 3 Is Responsible for Intestinal Tissue Damage during Mesenteric IschemiaAbhigyan Satyam, Lakshmi Kannan, Naoya Matsumoto, et al.
Nature Communications|June 7, 2020
Systemic lupus erythematosus favors the generation of IL-17 producing double negative T cellsHao Li, Iannis E Adamopoulos, Vaishali R Moulton, et al.
Pageof 6

Showing results (31-40 of 51) with videos related to

Sort By:
Pageof 6
Nature Communications|January 29, 2024
CaMK4 controls follicular helper T cell expansion and function during normal and autoimmune T-dependent B cell responsesMarc Scherlinger, Hao Li, Wenliang Pan, et al.
Shock (Augusta, Ga.)|April 19, 2019
Complement Deposition on the Surface of RBC After Trauma Serves a Biomarker of Moderate Trauma Severity: A Prospective StudyAbhigyan Satyam, Kostas Andreo, Peter H Lapchak, et al.
Clinical Immunology (Orlando, Fla.)|July 12, 2021
Complement activation and increased expression of Syk, mucin-1 and CaMK4 in kidneys of patients with COVID-19Simin Jamaly, Maria G Tsokos, Rhea Bhargava, et al.
Journal of Immunology (Baltimore, Md. : 1950)|September 8, 2017
IL-17A Produced by Innate Lymphoid Cells Is Essential for Intestinal Ischemia-Reperfusion InjuryMayya Geha, Maria G Tsokos, Robin E Bosse, et al.
The Journal of Clinical Investigation|May 6, 2021
IL-23 reshapes kidney resident cell metabolism and promotes local kidney inflammationHao Li, Maria G Tsokos, Rhea Bhargava, et al.
JCI Insight|March 30, 2021
Aberrantly glycosylated IgG elicits pathogenic signaling in podocytes and signifies lupus nephritisRhea Bhargava, Sylvain Lehoux, Kayaho Maeda, et al.
Science Translational Medicine|February 19, 2025
Gluconolactone restores immune regulation and alleviates skin inflammation in lupus-prone mice and in patients with cutaneous lupusWei Li, Antonios G A Kolios, Wenliang Pan, et al.
Science Advances|June 15, 2022
CD38 reduces mitochondrial fitness and cytotoxic T cell response against viral infection in lupus patients by suppressing mitophagyPing-Min Chen, Eri Katsuyama, Abhigyan Satyam, et al.
Journal of Immunology (Baltimore, Md. : 1950)|December 4, 2016
Intracellular Activation of Complement 3 Is Responsible for Intestinal Tissue Damage during Mesenteric IschemiaAbhigyan Satyam, Lakshmi Kannan, Naoya Matsumoto, et al.
Nature Communications|June 7, 2020
Systemic lupus erythematosus favors the generation of IL-17 producing double negative T cellsHao Li, Iannis E Adamopoulos, Vaishali R Moulton, et al.
Pageof 6