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Cancer Letters|November 17, 2018
Mitosis perturbation by MASTL depletion impairs the viability of thyroid tumor cellsElena Cetti, Tiziana Di Marco, Giuseppe Mauro, et al.
Endocrine-Related Cancer|June 22, 2011
Functional characterization of the MTC-associated germline RET-K666E mutation: evidence of oncogenic potential enhanced by the G691S polymorphismMaria Grazia Borrello, Antonella Aiello, Bernard Peissel, et al.
Frontiers in Endocrinology|October 23, 2023
Genomic and transcriptomic analyses of thyroid cancers identify DICER1 somatic mutations in adult follicular-patterned RAS-like tumorsEmanuela Minna, Andrea Devecchi, Federico Pistore, et al.
The Journal of Clinical Endocrinology and Metabolism|November 27, 2013
Synergistic cooperation between sunitinib and cisplatin promotes apoptotic cell death in human medullary thyroid cancerAlessia Lopergolo, Valentina Nicolini, Enrica Favini, et al.
Biology|October 23, 2021
Senescent Thyrocytes, Similarly to Thyroid Tumor Cells, Elicit M2-like Macrophage Polarization In VivoMara Mazzoni, Giuseppe Mauro, Lucia Minoli, et al.
Endocrine-Related Cancer|September 23, 2011
Evidence of oncogene-induced senescence in thyroid carcinogenesisMaria Grazia Vizioli, Patricia A Possik, Eva Tarantino, et al.
Journal of Experimental & Clinical Cancer Research : CR|May 23, 2019
Senescent thyrocytes and thyroid tumor cells induce M2-like macrophage polarization of human monocytes via a PGE2-dependent mechanismMara Mazzoni, Giuseppe Mauro, Marco Erreni, et al.
Thyroid : Official Journal of the American Thyroid Association|December 18, 2012
Evidence of a low prevalence of RAS mutations in a large medullary thyroid cancer seriesRaffaele Ciampi, Caterina Mian, Laura Fugazzola, et al.
Proceedings of the National Academy of Sciences of the United States of America|October 6, 2005
Induction of a proinflammatory program in normal human thyrocytes by the RET/PTC1 oncogeneMaria Grazia Borrello, Luisella Alberti, Andrew Fischer, et al.
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