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Nature Communications
|
February 25, 2025
KAT6B overexpression rescues embryonic lethality in homozygous null KAT6A mice restoring vitality and normal lifespan
Maria I Bergamasco, Yuqing Yang, Alexandra L Garnham, et al.
Development (Cambridge, England)
|
June 2, 2026
The roles of the acetyltransferase domains of the KAT6A and KAT6B in vivo
Tim Thomas, Shezlie Malelang, Yuqing Yang, et al.
Developmental Biology
|
January 20, 2025
Loss of KAT6B causes premature ossification and promotes osteoblast differentiation during development
Maria I Bergamasco, Jacqueline M Ogier, Alexandra L Garnham, et al.
Plos Genetics
|
May 4, 2026
KAT6A is essential for developmental control gene expression in neural stem and progenitor cells
Anne K Voss, Samantha Eccles, Johannes Wichmann, et al.
Life Science Alliance
|
November 13, 2024
KAT6B is required for histone 3 lysine 9 acetylation and SOX gene expression in the developing brain
Maria I Bergamasco, Waruni Abeysekera, Alexandra L Garnham, et al.
Cell Death and Differentiation
|
April 1, 2022
Loss of TAF8 causes TFIID dysfunction and p53-mediated apoptotic neuronal cell death
Farrah El-Saafin, Maria I Bergamasco, Yunshun Chen, et al.
Life Science Alliance
|
February 17, 2026
Acetyl-carnitine improves hyperactivity and learning deficits in <i>KAT6A</i> haploinsufficient mice
Samantha Eccles, Hannah K Vanyai, Maria I Bergamasco, et al.
The Journal of Clinical Investigation
|
April 1, 2024
Increasing histone acetylation improves sociability and restores learning and memory in KAT6B-haploinsufficient mice
Maria I Bergamasco, Hannah K Vanyai, Alexandra L Garnham, et al.
Development (Cambridge, England)
|
March 6, 2024
ING4 and ING5 are essential for histone H3 lysine 14 acetylation and epicardial cell lineage development
Sophia Y Y Mah, Hannah K Vanyai, Connie S N Li-Wai-Suen, et al.
Plos Genetics
|
October 15, 2024
Loss of PHF6 causes spontaneous seizures, enlarged brain ventricles and altered transcription in the cortex of a mouse model of the Börjeson-Forssman-Lehmann intellectual disability syndrome
Helen M McRae, Melody P Y Leong, Maria I Bergamasco, et al.
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of 2
Search research articles
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Showing results (1-10 of 14) with videos related to
Sort By:
Page
of 2
Nature Communications
|
February 25, 2025
KAT6B overexpression rescues embryonic lethality in homozygous null KAT6A mice restoring vitality and normal lifespan
Maria I Bergamasco, Yuqing Yang, Alexandra L Garnham, et al.
Development (Cambridge, England)
|
June 2, 2026
The roles of the acetyltransferase domains of the KAT6A and KAT6B in vivo
Tim Thomas, Shezlie Malelang, Yuqing Yang, et al.
Developmental Biology
|
January 20, 2025
Loss of KAT6B causes premature ossification and promotes osteoblast differentiation during development
Maria I Bergamasco, Jacqueline M Ogier, Alexandra L Garnham, et al.
Plos Genetics
|
May 4, 2026
KAT6A is essential for developmental control gene expression in neural stem and progenitor cells
Anne K Voss, Samantha Eccles, Johannes Wichmann, et al.
Life Science Alliance
|
November 13, 2024
KAT6B is required for histone 3 lysine 9 acetylation and SOX gene expression in the developing brain
Maria I Bergamasco, Waruni Abeysekera, Alexandra L Garnham, et al.
Cell Death and Differentiation
|
April 1, 2022
Loss of TAF8 causes TFIID dysfunction and p53-mediated apoptotic neuronal cell death
Farrah El-Saafin, Maria I Bergamasco, Yunshun Chen, et al.
Life Science Alliance
|
February 17, 2026
Acetyl-carnitine improves hyperactivity and learning deficits in <i>KAT6A</i> haploinsufficient mice
Samantha Eccles, Hannah K Vanyai, Maria I Bergamasco, et al.
The Journal of Clinical Investigation
|
April 1, 2024
Increasing histone acetylation improves sociability and restores learning and memory in KAT6B-haploinsufficient mice
Maria I Bergamasco, Hannah K Vanyai, Alexandra L Garnham, et al.
Development (Cambridge, England)
|
March 6, 2024
ING4 and ING5 are essential for histone H3 lysine 14 acetylation and epicardial cell lineage development
Sophia Y Y Mah, Hannah K Vanyai, Connie S N Li-Wai-Suen, et al.
Plos Genetics
|
October 15, 2024
Loss of PHF6 causes spontaneous seizures, enlarged brain ventricles and altered transcription in the cortex of a mouse model of the Börjeson-Forssman-Lehmann intellectual disability syndrome
Helen M McRae, Melody P Y Leong, Maria I Bergamasco, et al.
Page
of 2