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Metabolism: Clinical and Experimental|February 2, 2024
Intermittent fasting protects β-cell identity and function in a type-2 diabetes modelSumit Patel, Zihan Yan, Maria S RemediMedcomm|February 12, 2026
Loss of Beta-Cell Identity and Function as a Mechanism of Secondary Failure of Sulfonylurea Therapy in DiabetesSumit Patel, Zihan Yan, Maria S RemediDiabetes|February 23, 2022
ATP-Sensitive Potassium Channels in Hyperinsulinism and Type 2 Diabetes: Inconvenient Paradox or New Paradigm?Colin G Nichols, Nathaniel W York, Maria S RemediThe Journal of General Physiology|December 14, 2016
Diabetes induced by gain-of-function mutations in the Kir6.1 subunit of the KATP channelMaria S Remedi, Jonathan B Friedman, Colin G NicholsThe Journal of Clinical Investigation|November 16, 2020
Preferential Gq signaling in diabetes: an electrical switch in incretin action and in diabetes progression?Colin G Nichols, Nathaniel W York, Maria S RemediCell Metabolism|April 22, 2014
Pancreatic β cell dedifferentiation in diabetes and redifferentiation following insulin therapyZhiyu Wang, Nathaniel W York, Colin G Nichols, et al.Pediatric Diabetes|January 24, 2017
Sulfonylurea challenge test in subjects diagnosed with type 1 diabetes mellitusMaria S Remedi, Mareen Thomas, Colin G Nichols, et al.Proceedings of the National Academy of Sciences of the United States of America|November 4, 2021
Cognitive deficits and impaired hippocampal long-term potentiation in KATP-induced DEND syndromeShaul Yahil, David F Wozniak, Zihan Yan, et al.Journal of the American Heart Association|August 27, 2013
Hypotension due to Kir6.1 gain-of-function in vascular smooth muscleAnlong Li, Russell H Knutsen, Haixia Zhang, et al.Plos One|February 18, 2022
SGLT2 inhibitors therapy protects glucotoxicity-induced β-cell failure in a mouse model of human KATP-induced diabetes through mitigation of oxidative and ER stressZeenat A Shyr, Zihan Yan, Alessandro Ustione, et al.Pageof 7