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Diabetes, Obesity & Metabolism|July 2, 2021
High-fat diet prevents the development of autoimmune diabetes in NOD miceAmy L Clark, Zihan Yan, Sophia X Chen, et al.Islets|December 2, 2022
Genome-edited zebrafish model of ABCC8 loss-of-function diseaseJennifer M Ikle, Robert C Tryon, Soma S Singareddy, et al.Diabetes|March 16, 2022
Genetic Reduction of Glucose Metabolism Preserves Functional β-Cell Mass in KATP-Induced Neonatal DiabetesZihan Yan, Manuela Fortunato, Zeenat A Shyr, et al.Journal of the American Heart Association|December 14, 2022
A Unique High-Output Cardiac Hypertrophy Phenotype Arising From Low Systemic Vascular Resistance in Cantu SyndromeGautam K Singh, Conor McClenaghan, Manish Aggarwal, et al.Diabetologia|September 24, 2025
Restoration of pancreatic beta cell identity and autophagy in KATP-induced diabetes by intermittent fastingEsmeralda Castelblanco, Zeenat A Shyr, Irving Ramirez-Sotero, et al.The Journal of Clinical Endocrinology and Metabolism|December 13, 2007
The G53D mutation in Kir6.2 (KCNJ11) is associated with neonatal diabetes and motor dysfunction in adulthood that is improved with sulfonylurea therapyJoseph C Koster, Francesco Cadario, Cinzia Peruzzi, et al.Islets|December 7, 2018
In vivo monitoring of intracellular Ca2+ dynamics in the pancreatic β-cells of zebrafish embryosReka Lorincz, Christopher H Emfinger, Andrea Walcher, et al.American Journal of Physiology. Endocrinology and Metabolism|September 19, 2018
Contribution of systemic inflammation to permanence of KATP-induced neonatal diabetes in miceChristopher H Emfinger, Zihan Yan, Alecia Welscher, et al.Pediatric Diabetes|August 7, 2009
Successful sulfonylurea treatment of an insulin-naïve neonate with diabetes mellitus due to a KCNJ11 mutationJennifer A Wambach, Bess A Marshall, Joseph C Koster, et al.Journal of Molecular and Cellular Cardiology|July 17, 2012
Cardiac specific ATP-sensitive K+ channel (KATP) overexpression results in embryonic lethalityAmir Toib, Hai Xia Zhang, Thomas J Broekelmann, et al.Pageof 7