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Journal of the American Heart Association|August 27, 2013
Hypotension due to Kir6.1 gain-of-function in vascular smooth muscleAnlong Li, Russell H Knutsen, Haixia Zhang, et al.JCI Insight|February 2, 2021
Complex consequences of Cantu syndrome SUR2 variant R1154Q in genetically modified miceHaixia Zhang, Alex Hanson, Tobias Scherf de Almeida, et al.Physiological Reports|June 5, 2019
Beta-cell excitability and excitability-driven diabetes in adult Zebrafish isletsChristopher H Emfinger, Réka Lőrincz, Yixi Wang, et al.The Journal of Clinical Investigation|November 8, 2016
Rpl13a small nucleolar RNAs regulate systemic glucose metabolismJiyeon Lee, Alexis N Harris, Christopher L Holley, et al.American Journal of Medical Genetics. Part C, Seminars in Medical Genetics|December 13, 2019
Cantú syndrome: Findings from 74 patients in the International Cantú Syndrome RegistryDorothy K Grange, Helen I Roessler, Conor McClenaghan, et al.Cell Metabolism|January 12, 2023
Palmitoylation couples insulin hypersecretion with β cell failure in diabetesGuifang Dong, Sangeeta Adak, George Spyropoulos, et al.JCI Insight|May 2, 2023
KATP channels are necessary for glucose-dependent increases in amyloid-β and Alzheimer's disease-related pathologyJohn Grizzanti, William R Moritz, Morgan C Pait, et al.Nature Communications|October 3, 2019
ABCC9-related Intellectual disability Myopathy Syndrome is a KATP channelopathy with loss-of-function mutations in ABCC9Marie F Smeland, Conor McClenaghan, Helen I Roessler, et al.Pageof 6