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Maria Secrier

Showing results (31-40 of 47) with videos related to

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Science Signaling|April 14, 2026
Brain-derived neurotrophic factor coordinates neuron-intrinsic programs to enhance axonal regeneration in human motor neuronsJose Norberto S Vargas, Anna-Leigh Brown, Kai Sun, et al.
International Journal of Cancer|May 4, 2019
Transcriptomic profiling reveals three molecular phenotypes of adenocarcinoma at the gastroesophageal junctionJan Bornschein, Lorenz Wernisch, Maria Secrier, et al.
Cancers|July 24, 2021
Genomic Analysis of Response to Neoadjuvant Chemotherapy in Esophageal AdenocarcinomaFereshteh Izadi, Benjamin P Sharpe, Stella P Breininger, et al.
Nature Medicine|April 16, 2026
Integrated epidemiological and molecular data inform the relationship between precancer and cancer states of esophageal adenocarcinomaShahriar A Zamani, Lianlian Wu, Emily L Black, et al.
Nature Communications|August 1, 2018
Organoid cultures recapitulate esophageal adenocarcinoma heterogeneity providing a model for clonality studies and precision therapeuticsXiaodun Li, Hayley E Francies, Maria Secrier, et al.
Biorxiv : the Preprint Server for Biology|February 5, 2024
TDP-43 loss induces extensive cryptic polyadenylation in ALS/FTDSam Bryce-Smith, Anna-Leigh Brown, Puja R Mehta, et al.
Nature Genetics|September 6, 2016
Mutational signatures in esophageal adenocarcinoma define etiologically distinct subgroups with therapeutic relevanceMaria Secrier, Xiaodun Li, Nadeera de Silva, et al.
Nature Genetics|February 1, 2017
Corrigendum: Mutational signatures in esophageal adenocarcinoma define etiologically distinct subgroups with therapeutic relevanceMaria Secrier, Xiaodun Li, Nadeera de Silva, et al.
Nature Genetics|January 8, 2020
Genomic evidence supports a clonal diaspora model for metastases of esophageal adenocarcinomaAyesha Noorani, Xiaodun Li, Martin Goddard, et al.
Nature Neuroscience|October 21, 2025
TDP-43 loss induces cryptic polyadenylation in ALS/FTDSam Bryce-Smith, Anna-Leigh Brown, Max Z Y J Chien, et al.
Pageof 5

Showing results (31-40 of 47) with videos related to

Sort By:
Pageof 5
Science Signaling|April 14, 2026
Brain-derived neurotrophic factor coordinates neuron-intrinsic programs to enhance axonal regeneration in human motor neuronsJose Norberto S Vargas, Anna-Leigh Brown, Kai Sun, et al.
International Journal of Cancer|May 4, 2019
Transcriptomic profiling reveals three molecular phenotypes of adenocarcinoma at the gastroesophageal junctionJan Bornschein, Lorenz Wernisch, Maria Secrier, et al.
Cancers|July 24, 2021
Genomic Analysis of Response to Neoadjuvant Chemotherapy in Esophageal AdenocarcinomaFereshteh Izadi, Benjamin P Sharpe, Stella P Breininger, et al.
Nature Medicine|April 16, 2026
Integrated epidemiological and molecular data inform the relationship between precancer and cancer states of esophageal adenocarcinomaShahriar A Zamani, Lianlian Wu, Emily L Black, et al.
Nature Communications|August 1, 2018
Organoid cultures recapitulate esophageal adenocarcinoma heterogeneity providing a model for clonality studies and precision therapeuticsXiaodun Li, Hayley E Francies, Maria Secrier, et al.
Biorxiv : the Preprint Server for Biology|February 5, 2024
TDP-43 loss induces extensive cryptic polyadenylation in ALS/FTDSam Bryce-Smith, Anna-Leigh Brown, Puja R Mehta, et al.
Nature Genetics|September 6, 2016
Mutational signatures in esophageal adenocarcinoma define etiologically distinct subgroups with therapeutic relevanceMaria Secrier, Xiaodun Li, Nadeera de Silva, et al.
Nature Genetics|February 1, 2017
Corrigendum: Mutational signatures in esophageal adenocarcinoma define etiologically distinct subgroups with therapeutic relevanceMaria Secrier, Xiaodun Li, Nadeera de Silva, et al.
Nature Genetics|January 8, 2020
Genomic evidence supports a clonal diaspora model for metastases of esophageal adenocarcinomaAyesha Noorani, Xiaodun Li, Martin Goddard, et al.
Nature Neuroscience|October 21, 2025
TDP-43 loss induces cryptic polyadenylation in ALS/FTDSam Bryce-Smith, Anna-Leigh Brown, Max Z Y J Chien, et al.
Pageof 5