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Circulation. Heart Failure|August 19, 2021
Endothelial-Mesenchymal Transition in Heart Failure With a Preserved Ejection Fraction: Insights Into the Cardiorenal SyndromeMaría Valero-Muñoz, Albin Oh, Elizabeth Faudoa, et al.Arthritis Research & Therapy|November 11, 2022
Periostin overexpression in scleroderma cardiac tissue and its utility as a marker for disease complicationsFatima El-Adili, Justin K Lui, Mortada Najem, et al.The American Journal of Cardiology|July 17, 2007
Usefulness of the aldosterone synthase gene polymorphism C-344-T to predict cardiac remodeling in African-Americans versus non-African-Americans with chronic systolic heart failureAndreia Biolo, Tania Chao, Toni-Ann S Duhaney, et al.The Journal of Biological Chemistry|October 16, 2009
Cardiac-specific deletion of LKB1 leads to hypertrophy and dysfunctionYasumasa Ikeda, Kaori Sato, David R Pimentel, et al.Journal of the American Heart Association|August 27, 2019
Cardiovascular Manifestations of Systemic Sclerosis: A Danish Nationwide Cohort StudySheraz A Butt, Jørgen L Jeppesen, Christian Torp-Pedersen, et al.Circulation|February 3, 2005
Mineralocorticoid receptor inhibition ameliorates the transition to myocardial failure and decreases oxidative stress and inflammation in mice with chronic pressure overloadGabriela M Kuster, Eugene Kotlyar, Mary K Rude, et al.Circulation. Heart Failure|August 24, 2014
Effects of adiponectin on calcium-handling proteins in heart failure with preserved ejection fractionKomei Tanaka, Richard M Wilson, Eric E Essick, et al.The American Journal of Pathology|January 13, 2011
Evidence for a functional role of the molecular chaperone clusterin in amyloidotic cardiomyopathyMichael J Greene, Flora Sam, Pamela T Soo Hoo, et al.American Journal of Physiology. Heart and Circulatory Physiology|June 14, 2011
Adiponectin mediates cardioprotection in oxidative stress-induced cardiac myocyte remodelingEric E Essick, Noriyuki Ouchi, Richard M Wilson, et al.Journal of Molecular and Cellular Cardiology|May 15, 2007
Adiponectin protects against the development of systolic dysfunction following myocardial infarctionRei Shibata, Yasuhiro Izumiya, Kaori Sato, et al.Pageof 7