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Marie Nollet

Showing results (11-20 of 15) with videos related to

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F&S Science|May 13, 2022
Soluble CD146 is increased in preeclampsia and interacts with galectin-1 to regulate trophoblast migration through VEGFR2 receptorSylvie Bouvier, Waël Traboulsi, Sandra M Blois, et al.
EMBO Molecular Medicine|April 2, 2015
Defects in mitophagy promote redox-driven metabolic syndrome in the absence of TP53INP1Marion Seillier, Laurent Pouyet, Prudence N'Guessan, et al.
Autophagy|December 9, 2014
Autophagy in osteoblasts is involved in mineralization and bone homeostasisMarie Nollet, Sabine Santucci-Darmanin, Véronique Breuil, et al.
Theranostics|May 4, 2018
Soluble CD146 is a predictive marker of pejorative evolution and of sunitinib efficacy in clear cell renal cell carcinomaMaeva Dufies, Marie Nollet, Damien Ambrosetti, et al.
Molecular Oncology|February 17, 2023
MET exon 14 skipping mutation is a hepatocyte growth factor (HGF)-dependent oncogenic driver in vitro and in humanised HGF knock-in miceMarie Fernandes, Brynna Hoggard, Philippe Jamme, et al.
Pageof 2

Showing results (11-20 of 15) with videos related to

Sort By:
Pageof 2
You have reached the last page of results.This site can display upto 15 results.
F&S Science|May 13, 2022
Soluble CD146 is increased in preeclampsia and interacts with galectin-1 to regulate trophoblast migration through VEGFR2 receptorSylvie Bouvier, Waël Traboulsi, Sandra M Blois, et al.
EMBO Molecular Medicine|April 2, 2015
Defects in mitophagy promote redox-driven metabolic syndrome in the absence of TP53INP1Marion Seillier, Laurent Pouyet, Prudence N'Guessan, et al.
Autophagy|December 9, 2014
Autophagy in osteoblasts is involved in mineralization and bone homeostasisMarie Nollet, Sabine Santucci-Darmanin, Véronique Breuil, et al.
Theranostics|May 4, 2018
Soluble CD146 is a predictive marker of pejorative evolution and of sunitinib efficacy in clear cell renal cell carcinomaMaeva Dufies, Marie Nollet, Damien Ambrosetti, et al.
Molecular Oncology|February 17, 2023
MET exon 14 skipping mutation is a hepatocyte growth factor (HGF)-dependent oncogenic driver in vitro and in humanised HGF knock-in miceMarie Fernandes, Brynna Hoggard, Philippe Jamme, et al.
Pageof 2