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Marie-Christine Galas

Showing results (1-10 of 50) with videos related to

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The Journal of Biological Chemistry|August 23, 2015
Amyloid Precursor Protein (APP) Metabolites APP Intracellular Fragment (AICD), Aβ42, and Tau in Nuclear RolesGerhard Multhaup, Otmar Huber, Luc Buée, et al.
Methods in Molecular Biology (Clifton, N.J.)|December 16, 2016
In Vivo Hyperthermic Stress Model: An Easy Tool to Study the Effects of Oxidative Stress on Neuronal Tau Functionality in Mouse BrainAlban Chauderlier, Lucie Delattre, Luc Buée, et al.
Advances in Experimental Medicine and Biology|February 26, 2020
Emerging Connections Between Tau and Nucleic AcidsMarie-Christine Galas, Eliette Bonnefoy, Luc Buee, et al.
Neuroscience Letters|January 1, 2004
Chronic intoxication with 3-nitropropionic acid in rats induces the loss of striatal dopamine terminals without affecting nigral cell viabilityDavid Blum, Marie-Christine Galas, Laetitia Cuvelier, et al.
Brain Pathology (Zurich, Switzerland)|April 22, 2024
Cell redistribution of G quadruplex-structured DNA is associated with morphological changes of nuclei and nucleoli in neurons during tau pathology progressionThomas Comptdaer, Meryem Tardivel, Claire Schirmer, et al.
The Lancet. Neurology|July 10, 2003
Adenosine receptors and Huntington's disease: implications for pathogenesis and therapeuticsDavid Blum, Raphaël Hourez, Marie-Christine Galas, et al.
Neurobiology of Disease|September 25, 2002
Striatal and cortical neurochemical changes induced by chronic metabolic compromise in the 3-nitropropionic model of Huntington's diseaseDavid Blum, Marie-Christine Galas, David Gall, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|October 22, 2002
The adenosine A1 receptor agonist adenosine amine congener exerts a neuroprotective effect against the development of striatal lesions and motor impairments in the 3-nitropropionic acid model of neurotoxicityDavid Blum, David Gall, Marie-Christine Galas, et al.
Journal of Alzheimer'S Disease : JAD|January 29, 2019
Lipidized Prolactin-Releasing Peptide Agonist Attenuates Hypothermia-Induced Tau Hyperphosphorylation in NeuronsVeronika Pražienková, Claire Schirmer, Martina Holubová, et al.
The Journal of Biological Chemistry|May 6, 2006
The peptidylprolyl cis/trans-isomerase Pin1 modulates stress-induced dephosphorylation of Tau in neurons. Implication in a pathological mechanism related to Alzheimer diseaseMarie-Christine Galas, Pierre Dourlen, Séverine Bégard, et al.
Pageof 5

Showing results (1-10 of 50) with videos related to

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Pageof 5
The Journal of Biological Chemistry|August 23, 2015
Amyloid Precursor Protein (APP) Metabolites APP Intracellular Fragment (AICD), Aβ42, and Tau in Nuclear RolesGerhard Multhaup, Otmar Huber, Luc Buée, et al.
Methods in Molecular Biology (Clifton, N.J.)|December 16, 2016
In Vivo Hyperthermic Stress Model: An Easy Tool to Study the Effects of Oxidative Stress on Neuronal Tau Functionality in Mouse BrainAlban Chauderlier, Lucie Delattre, Luc Buée, et al.
Advances in Experimental Medicine and Biology|February 26, 2020
Emerging Connections Between Tau and Nucleic AcidsMarie-Christine Galas, Eliette Bonnefoy, Luc Buee, et al.
Neuroscience Letters|January 1, 2004
Chronic intoxication with 3-nitropropionic acid in rats induces the loss of striatal dopamine terminals without affecting nigral cell viabilityDavid Blum, Marie-Christine Galas, Laetitia Cuvelier, et al.
Brain Pathology (Zurich, Switzerland)|April 22, 2024
Cell redistribution of G quadruplex-structured DNA is associated with morphological changes of nuclei and nucleoli in neurons during tau pathology progressionThomas Comptdaer, Meryem Tardivel, Claire Schirmer, et al.
The Lancet. Neurology|July 10, 2003
Adenosine receptors and Huntington's disease: implications for pathogenesis and therapeuticsDavid Blum, Raphaël Hourez, Marie-Christine Galas, et al.
Neurobiology of Disease|September 25, 2002
Striatal and cortical neurochemical changes induced by chronic metabolic compromise in the 3-nitropropionic model of Huntington's diseaseDavid Blum, Marie-Christine Galas, David Gall, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|October 22, 2002
The adenosine A1 receptor agonist adenosine amine congener exerts a neuroprotective effect against the development of striatal lesions and motor impairments in the 3-nitropropionic acid model of neurotoxicityDavid Blum, David Gall, Marie-Christine Galas, et al.
Journal of Alzheimer'S Disease : JAD|January 29, 2019
Lipidized Prolactin-Releasing Peptide Agonist Attenuates Hypothermia-Induced Tau Hyperphosphorylation in NeuronsVeronika Pražienková, Claire Schirmer, Martina Holubová, et al.
The Journal of Biological Chemistry|May 6, 2006
The peptidylprolyl cis/trans-isomerase Pin1 modulates stress-induced dephosphorylation of Tau in neurons. Implication in a pathological mechanism related to Alzheimer diseaseMarie-Christine Galas, Pierre Dourlen, Séverine Bégard, et al.
Pageof 5