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Current Opinion in Cardiology|March 5, 2013
'State-of-the-heart' of cardiac laminopathiesMarie-Elodie Cattin, Antoine Muchir, Gisèle Bonne
Current Opinion in Pharmacology|July 23, 2018
Filling the drug discovery gap: is high-content screening the missing link?Thierry Dorval, Benjamin Chanrion, Marie-Elodie Cattin, et al.
Neuromuscular Disorders : NMD|June 12, 2016
Mutation in lamin A/C sensitizes the myocardium to exercise-induced mechanical stress but has no effect on skeletal muscles in mouseMarie-Elodie Cattin, Arnaud Ferry, Alban Vignaud, et al.
Stem Cells (Dayton, Ohio)|December 29, 2017
In Vitro Modeling of Congenital Heart Defects Associated with an NKX2-5 Mutation Revealed a Dysregulation in BMP/Notch-Mediated SignalingAbeer F Zakariyah, Rashida F Rajgara, Ellias Horner, et al.
JCI Insight|January 2, 2024
hsa-miR-548v controls the viscoelastic properties of human cardiomyocytes and improves their relaxation ratesEva Vermersch, Salomé Neuvendel, Charlène Jouve, et al.
The Journal of Biological Chemistry|September 12, 2015
Deletion of MLIP (muscle-enriched A-type lamin-interacting protein) leads to cardiac hyperactivation of Akt/mammalian target of rapamycin (mTOR) and impaired cardiac adaptationMarie-Elodie Cattin, Jessica Wang, Jonathan J Weldrick, et al.
Human Molecular Genetics|November 18, 2011
DelK32-lamin A/C has abnormal location and induces incomplete tissue maturation and severe metabolic defects leading to premature deathAnne T Bertrand, Laure Renou, Aurélie Papadopoulos, et al.
The Journal of Biological Chemistry|November 4, 2018
Expression of murine muscle-enriched A-type lamin-interacting protein (MLIP) is regulated by tissue-specific alternative transcription start sitesMarie-Elodie Cattin, Shelley A Deeke, Sarah A Dick, et al.
Human Molecular Genetics|April 12, 2013
Heterozygous LmnadelK32 mice develop dilated cardiomyopathy through a combined pathomechanism of haploinsufficiency and peptide toxicityMarie-Elodie Cattin, Anne T Bertrand, Saskia Schlossarek, et al.
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