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Marie-Lune Simard

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The Journal of Pathology|April 17, 2018
A novel histochemistry assay to assess and quantify focal cytochrome c oxidase deficiencyMarie-Lune Simard, Arnaud Mourier, Laura C Greaves, et al.
Experimental & Translational Stroke Medicine|December 22, 2010
Dynamics of neuroinflammation in the macrosphere model of arterio-arterial embolic focal ischemia: an approximation to human stroke patternsMaureen Walberer, Maria A Rueger, Marie-Lune Simard, et al.
Neuroscience Research|August 28, 2007
LDL receptor deficiency results in decreased cell proliferation and presynaptic bouton density in the murine hippocampusMonique Mulder, Guido Koopmans, Guido Wassink, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|May 7, 2010
Noninvasive imaging of endogenous neural stem cell mobilization in vivo using positron emission tomographyMaria Adele Rueger, Heiko Backes, Maureen Walberer, et al.
Cell Reports|September 15, 2016
A Phenotype-Driven Approach to Generate Mouse Models with Pathogenic mtDNA Mutations Causing Mitochondrial DiseaseJohanna H K Kauppila, Holly L Baines, Ana Bratic, et al.
Annals of Neurology|April 25, 2006
Natalizumab effects on immune cell responses in multiple sclerosisMasaaki Niino, Caroline Bodner, Marie-Lune Simard, et al.
Plos Genetics|May 9, 2022
Mice lacking the mitochondrial exonuclease MGME1 develop inflammatory kidney disease with glomerular dysfunctionDusanka Milenkovic, Adrián Sanz-Moreno, Julia Calzada-Wack, et al.
Nature Communications|March 25, 2018
Mice lacking the mitochondrial exonuclease MGME1 accumulate mtDNA deletions without developing progeriaStanka Matic, Min Jiang, Thomas J Nicholls, et al.
Nature Medicine|September 26, 2018
Genome editing in mitochondria corrects a pathogenic mtDNA mutation in vivoPayam A Gammage, Carlo Viscomi, Marie-Lune Simard, et al.
Cell|February 24, 2023
Cell lineage-specific mitochondrial resilience during mammalian organogenesisStephen P Burr, Florian Klimm, Angelos Glynos, et al.
Pageof 1

Showing results (1-10 of 10) with videos related to

Sort By:
Pageof 1
The Journal of Pathology|April 17, 2018
A novel histochemistry assay to assess and quantify focal cytochrome c oxidase deficiencyMarie-Lune Simard, Arnaud Mourier, Laura C Greaves, et al.
Experimental & Translational Stroke Medicine|December 22, 2010
Dynamics of neuroinflammation in the macrosphere model of arterio-arterial embolic focal ischemia: an approximation to human stroke patternsMaureen Walberer, Maria A Rueger, Marie-Lune Simard, et al.
Neuroscience Research|August 28, 2007
LDL receptor deficiency results in decreased cell proliferation and presynaptic bouton density in the murine hippocampusMonique Mulder, Guido Koopmans, Guido Wassink, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|May 7, 2010
Noninvasive imaging of endogenous neural stem cell mobilization in vivo using positron emission tomographyMaria Adele Rueger, Heiko Backes, Maureen Walberer, et al.
Cell Reports|September 15, 2016
A Phenotype-Driven Approach to Generate Mouse Models with Pathogenic mtDNA Mutations Causing Mitochondrial DiseaseJohanna H K Kauppila, Holly L Baines, Ana Bratic, et al.
Annals of Neurology|April 25, 2006
Natalizumab effects on immune cell responses in multiple sclerosisMasaaki Niino, Caroline Bodner, Marie-Lune Simard, et al.
Plos Genetics|May 9, 2022
Mice lacking the mitochondrial exonuclease MGME1 develop inflammatory kidney disease with glomerular dysfunctionDusanka Milenkovic, Adrián Sanz-Moreno, Julia Calzada-Wack, et al.
Nature Communications|March 25, 2018
Mice lacking the mitochondrial exonuclease MGME1 accumulate mtDNA deletions without developing progeriaStanka Matic, Min Jiang, Thomas J Nicholls, et al.
Nature Medicine|September 26, 2018
Genome editing in mitochondria corrects a pathogenic mtDNA mutation in vivoPayam A Gammage, Carlo Viscomi, Marie-Lune Simard, et al.
Cell|February 24, 2023
Cell lineage-specific mitochondrial resilience during mammalian organogenesisStephen P Burr, Florian Klimm, Angelos Glynos, et al.
Pageof 1