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Proceedings of the National Academy of Sciences of the United States of America|March 9, 2005
Reduced intercellular coupling leads to paradoxical propagation across the Purkinje-ventricular junction and aberrant myocardial activationGregory E Morley, Stephan B Danik, Scott Bernstein, et al.Scientific Reports|February 15, 2020
Connexin43 expression in bone marrow derived cells contributes to the electrophysiological properties of cardiac scar tissueCarolina Vasquez, Valeria Mezzano, Newman Kessler, et al.Circulation Research|June 27, 2009
Novel pharmacophores of connexin43 based on the "RXP" series of Cx43-binding peptidesVandana Verma, Bjarne Due Larsen, Wanda Coombs, et al.JACC. Clinical Electrophysiology|January 26, 2019
Plakophilin-2 Truncation Variants in Patients Clinically Diagnosed With Catecholaminergic Polymorphic Ventricular Tachycardia and Decedents With Exercise-Associated Autopsy Negative Sudden Unexplained Death in the YoungDavid J Tester, Jaeger P Ackerman, John R Giudicessi, et al.Journal of Biomedicine & Biotechnology|May 15, 2010
Ordered assembly of the adhesive and electrochemical connections within newly formed intercalated disks in primary cultures of adult rat cardiomyocytesSarah B Geisler, Kathleen J Green, Lori L Isom, et al.Scientific Reports|July 8, 2020
Author Correction: Connexin43 expression in bone marrow derived cells contributes to the electrophysiological properties of cardiac scar tissueCarolina Vasquez, Valeria Mezzano, Newman Kessler, et al.The Journal of Clinical Investigation|October 9, 2014
PCP4 regulates Purkinje cell excitability and cardiac rhythmicityEugene E Kim, Akshay Shekhar, Jia Lu, et al.Circulation. Arrhythmia and Electrophysiology|January 3, 2023
Chronic Kidney Disease Induces Proarrhythmic RemodelingBenjamin M N King, Shana Mintz, Xianming Lin, et al.Circulation Research|October 23, 2004
Modulation of cardiac gap junction expression and arrhythmic susceptibilityStephan B Danik, Fangyu Liu, Jie Zhang, et al.The Journal of Cell Biology|February 10, 2016
Plakophilin-2 loss promotes TGF-β1/p38 MAPK-dependent fibrotic gene expression in cardiomyocytesAdi D Dubash, Chen Y Kam, Brian A Aguado, et al.Pageof 16