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Current Opinion in Cardiology|March 3, 2012
Cardiac connexins, mutations and arrhythmiasMario Delmar, Naomasa MakitaCirculation Journal : Official Journal of the Japanese Circulation Society|April 2, 2009
Phenotypic overlap of cardiac sodium channelopathies: individual-specific or mutation-specific?Naomasa MakitaCirculation Journal : Official Journal of the Japanese Circulation Society|December 6, 2007
Genetic polymorphisms and arrhythmia susceptibilityNaomasa Makita, Hiroyuki TsutsuiCirculation Journal : Official Journal of the Japanese Circulation Society|May 27, 2008
Absence of a trafficking defect in R1232W/T1620M, a double SCN5A mutant responsible for Brugada syndromeNaomasa Makita, Naoki Mochizuki, Hiroyuki TsutsuiPediatric Cardiology|March 13, 2012
Desmosome-ion channel interactions and their possible role in arrhythmogenic cardiomyopathyMario DelmarJournal of Arrhythmia|October 21, 2016
Inherited bradyarrhythmia: A diverse genetic backgroundTaisuke Ishikawa, Yukiomi Tsuji, Naomasa MakitaCardiovascular Research|December 20, 2011
Connexin43 regulates sodium current; ankyrin-G modulates gap junctions: the intercalated disc exchangerMario DelmarHeart Rhythm|April 3, 2007
Novel SCN5A mutation (Q55X) associated with age-dependent expression of Brugada syndrome presenting as neurally mediated syncopeNaomasa Makita, Naokata Sumitomo, Ichiro Watanabe, et al.Pediatrics International : Official Journal of the Japan Pediatric Society|May 26, 2020
Dynamic QT response to cold-water face immersion in long-QT syndrome type 3Kazuhiro Takahashi, Wataru Shimizu, Naomasa Makita, et al.Trends in Cardiovascular Medicine|March 25, 2014
Desmosomes and the sodium channel complex: implications for arrhythmogenic cardiomyopathy and Brugada syndromeMarina Cerrone, Mario DelmarPageof 22