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Mario Delmar

Showing results (101-110 of 122) with videos related to

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Neuron|February 4, 2018
Localized Myosin II Activity Regulates Assembly and Plasticity of the Axon Initial SegmentStephen L Berger, Alejandra Leo-Macias, Stephanie Yuen, et al.
Circulation|December 20, 2013
Missense mutations in plakophilin-2 cause sodium current deficit and associate with a Brugada syndrome phenotypeMarina Cerrone, Xianming Lin, Mingliang Zhang, et al.
Circulation. Arrhythmia and Electrophysiology|December 10, 2016
Scn2b Deletion in Mice Results in Ventricular and Atrial ArrhythmiasYangyang Bao, B Cicero Willis, Chad R Frasier, et al.
Journal of Cell Science|January 22, 2025
Meeting report - Alpine desmosome disease meeting 2024: advances and emerging topics in desmosomes and related diseasesJens Waschke, Masayuki Amagai, Christoph Becker, et al.
Biorxiv : the Preprint Server for Biology|November 24, 2025
Cardiomyocyte-specific plakophilin-2 loss is sufficient to induce aging and senescence of nonmyocytes. Relevance to arrhythmogenic cardiomyopathyGiorgia Bertoli, Kavya Phadke, Alessandro Cospito, et al.
Journal of the American Heart Association|April 28, 2026
Cardiomyocyte-Specific Plakophilin-2 Loss Is Sufficient to Induce Aging and Senescence of Nonmyocytes: Relevance to Arrhythmogenic CardiomyopathyGiorgia Bertoli, Kavya Phadke, Alessandro Cospito, et al.
The Journal of Clinical Investigation|February 23, 2021
Cx43 hemichannel microdomain signaling at the intercalated disc enhances cardiac excitabilityMaarten Aj De Smet, Alessio Lissoni, Timur Nezlobinsky, et al.
Heart Rhythm|November 22, 2011
Reduced heterogeneous expression of Cx43 results in decreased Nav1.5 expression and reduced sodium current that accounts for arrhythmia vulnerability in conditional Cx43 knockout miceJohn A Jansen, Maartje Noorman, Hassan Musa, et al.
Basic Research in Cardiology|April 6, 2013
Deletion of the last five C-terminal amino acid residues of connexin43 leads to lethal ventricular arrhythmias in mice without affecting coupling via gap junction channelsIndra Lübkemeier, Robert Pascal Requardt, Xianming Lin, et al.
Circulation|May 3, 2018
Mechanosensitive Gene Regulation by Myocardin-Related Transcription Factors Is Required for Cardiomyocyte Integrity in Load-Induced Ventricular HypertrophyMichael A Trembley, Pearl Quijada, Esperanza Agullo-Pascual, et al.
Pageof 13

Showing results (101-110 of 122) with videos related to

Sort By:
Pageof 13
Neuron|February 4, 2018
Localized Myosin II Activity Regulates Assembly and Plasticity of the Axon Initial SegmentStephen L Berger, Alejandra Leo-Macias, Stephanie Yuen, et al.
Circulation|December 20, 2013
Missense mutations in plakophilin-2 cause sodium current deficit and associate with a Brugada syndrome phenotypeMarina Cerrone, Xianming Lin, Mingliang Zhang, et al.
Circulation. Arrhythmia and Electrophysiology|December 10, 2016
Scn2b Deletion in Mice Results in Ventricular and Atrial ArrhythmiasYangyang Bao, B Cicero Willis, Chad R Frasier, et al.
Journal of Cell Science|January 22, 2025
Meeting report - Alpine desmosome disease meeting 2024: advances and emerging topics in desmosomes and related diseasesJens Waschke, Masayuki Amagai, Christoph Becker, et al.
Biorxiv : the Preprint Server for Biology|November 24, 2025
Cardiomyocyte-specific plakophilin-2 loss is sufficient to induce aging and senescence of nonmyocytes. Relevance to arrhythmogenic cardiomyopathyGiorgia Bertoli, Kavya Phadke, Alessandro Cospito, et al.
Journal of the American Heart Association|April 28, 2026
Cardiomyocyte-Specific Plakophilin-2 Loss Is Sufficient to Induce Aging and Senescence of Nonmyocytes: Relevance to Arrhythmogenic CardiomyopathyGiorgia Bertoli, Kavya Phadke, Alessandro Cospito, et al.
The Journal of Clinical Investigation|February 23, 2021
Cx43 hemichannel microdomain signaling at the intercalated disc enhances cardiac excitabilityMaarten Aj De Smet, Alessio Lissoni, Timur Nezlobinsky, et al.
Heart Rhythm|November 22, 2011
Reduced heterogeneous expression of Cx43 results in decreased Nav1.5 expression and reduced sodium current that accounts for arrhythmia vulnerability in conditional Cx43 knockout miceJohn A Jansen, Maartje Noorman, Hassan Musa, et al.
Basic Research in Cardiology|April 6, 2013
Deletion of the last five C-terminal amino acid residues of connexin43 leads to lethal ventricular arrhythmias in mice without affecting coupling via gap junction channelsIndra Lübkemeier, Robert Pascal Requardt, Xianming Lin, et al.
Circulation|May 3, 2018
Mechanosensitive Gene Regulation by Myocardin-Related Transcription Factors Is Required for Cardiomyocyte Integrity in Load-Induced Ventricular HypertrophyMichael A Trembley, Pearl Quijada, Esperanza Agullo-Pascual, et al.
Pageof 13