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Clinical and Vaccine Immunology : CVI|November 17, 2006
Borrelia burgdorferi spirochetes that harbor only a portion of the lp28-1 plasmid elicit antibody responses detectable with the C6 test for Lyme diseaseMonica E Embers, Gary P Wormser, Ira Schwartz, et al.Vaccine|June 1, 2011
A protein-based pneumococcal vaccine protects rhesus macaques from pneumonia after experimental infection with Streptococcus pneumoniaePhilippe Denoël, Mario T Philipp, Lara Doyle, et al.Infection and Immunity|August 13, 2008
Toll-like receptors: insights into their possible role in the pathogenesis of lyme neuroborreliosisAndrea L F Bernardino, Tereance A Myers, Xavier Alvarez, et al.Journal of Neuroinflammation|July 23, 2013
The Lyme disease spirochete Borrelia burgdorferi induces inflammation and apoptosis in cells from dorsal root gangliaGeeta Ramesh, Lenay Santana-Gould, Fiona M Inglis, et al.Journal of Clinical Microbiology|November 8, 2003
C6 test as an indicator of therapy outcome for patients with localized or disseminated lyme borreliosisMario T Philipp, Adriana R Marques, Paul T Fawcett, et al.Infection and Immunity|March 16, 2002
Autocrine and exocrine regulation of interleukin-10 production in THP-1 cells stimulated with Borrelia burgdorferi lipoproteinsGuillermo H Giambartolomei, Vida A Dennis, Barbara L Lasater, et al.Journal of Immunology (Baltimore, Md. : 1950)|September 24, 2004
Lipoproteins, not lipopolysaccharide, are the key mediators of the proinflammatory response elicited by heat-killed Brucella abortusGuillermo H Giambartolomei, Astrid Zwerdling, Juliana Cassataro, et al.Infection and Immunity|January 14, 2009
Live Borrelia burgdorferi spirochetes elicit inflammatory mediators from human monocytes via the Toll-like receptor signaling pathwayVida A Dennis, Saurabh Dixit, Shannon M O'Brien, et al.Journal of Neuroinflammation|February 17, 2017
Aprepitant limits in vivo neuroinflammatory responses in a rhesus model of Lyme neuroborreliosisAlejandra N Martinez, Amanda R Burmeister, Geeta Ramesh, et al.Plos Pathogens|December 10, 2009
Central role of the Holliday junction helicase RuvAB in vlsE recombination and infectivity of Borrelia burgdorferiTao Lin, Lihui Gao, Diane G Edmondson, et al.Pageof 7