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[Rinsho Ketsueki] the Japanese Journal of Clinical Hematology|September 10, 2020
[Mutant calreticulin and the molecular mechanisms in development of myeloproliferative neoplasms]Marito Araki[Rinsho Ketsueki] the Japanese Journal of Clinical Hematology|September 7, 2018
[Development of myeloproliferative neoplasms by mutant calreticulin: underlying mechanisms]Marito ArakiInternational Journal of Hematology|December 19, 2019
The role of calreticulin mutations in myeloproliferative neoplasmsMarito Araki, Norio KomatsuCancer Science|July 26, 2017
Novel molecular mechanism of cellular transformation by a mutant molecular chaperone in myeloproliferative neoplasmsMarito Araki, Norio Komatsu[Rinsho Ketsueki] the Japanese Journal of Clinical Hematology|January 17, 2017
Gene mutations in myeloproliferative neoplasmsMarito Araki, Soji Morishita, Norio KomatsuGenes & Development|October 1, 2005
A novel motif governs APC-dependent degradation of Drosophila ORC1 in vivoMarito Araki, Hongtao Yu, Maki AsanoCancer Science|May 29, 2020
Mechanism underlying the development of myeloproliferative neoplasms through mutant calreticulinYoko Edahiro, Marito Araki, Norio KomatsuInternational Journal of Hematology|May 5, 2017
Somatic mutations of calreticulin in myeloproliferative neoplasmsMisa Imai, Marito Araki, Norio KomatsuExpert Review of Hematology|April 18, 2019
Molecular features, prognosis, and novel treatment options for pediatric acute megakaryoblastic leukemiaFederico De Marchi, Marito Araki, Norio KomatsuBiochemical and Biophysical Research Communications|March 7, 2015
Platelet-derived growth factor receptors form complexes with neuropilin-1 during megakaryocytic differentiation of thrombopoietin-dependent UT-7/TPO cellsAkimichi Ohsaka, Satoko Hirota-Komatsu, Marito Araki, et al.Pageof 6