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Biomolecules|January 8, 2025
Bioactivation, Mutagenicity, DNA Damage, and Oxidative Stress Induced by 3,4-DimethylanilineMariam R Habil, Raúl A Salazar-González, Mark A Doll, et al.
Carcinogenesis|April 17, 2007
Functional characterization of single-nucleotide polymorphisms and haplotypes of human N-acetyltransferase 2Yu Zang, Mark A Doll, Shuang Zhao, et al.
Archives of Toxicology|September 16, 2022
N-acetyltransferase 2 acetylator genotype-dependent N-acetylation and toxicity of the arylamine carcinogen β-naphthylamine in cryopreserved human hepatocytesMariam R Habil, Raúl A Salazar-González, Mark A Doll, et al.
Archives of Toxicology|August 30, 2022
Differences in β-naphthylamine metabolism and toxicity in Chinese hamster ovary cell lines transfected with human CYP1A2 and NAT2*4, NAT2*5B or NAT2*7B N-acetyltransferase 2 haplotypesMariam R Habil, Raúl A Salazar-González, Mark A Doll, et al.
Pharmacogenetics and Genomics|February 1, 2007
Functional characterization of the A411T (L137F) and G364A (D122N) genetic polymorphisms in human N-acetyltransferase 2Yu Zang, Shuang Zhao, Mark A Doll, et al.
Chemical Research in Toxicology|February 27, 2009
The impact of NAT2 acetylator genotype on mutagenesis and DNA adducts from 2-amino-9H-pyrido[2,3-b]indoleRobert J Turesky, Jean Bendaly, Isil Yasa, et al.
Archives of Toxicology|May 4, 2023
Effect of N-acetyltransferase 2 genetic polymorphism on 4,4'-methylenebis(2-chloroaniline)-induced genotoxicity and oxidative stressMariam R Habil, Raúl A Salazar-González, Mark A Doll, et al.
Molecular Carcinogenesis|August 13, 2011
NATb/NAT1*4 promotes greater arylamine N-acetyltransferase 1 mediated DNA adducts and mutations than NATa/NAT1*4 following exposure to 4-aminobiphenylLori M Millner, Mark A Doll, Jian Cai, et al.
Investigative Ophthalmology & Visual Science|April 12, 2014
The role of endogenous epidermal growth factor receptor ligands in mediating corneal epithelial homeostasisJoanne L Peterson, Eric D Phelps, Mark A Doll, et al.
BMC Cancer|September 2, 2020
UBR-box containing protein, UBR5, is over-expressed in human lung adenocarcinoma and is a potential therapeutic targetKumar Saurabh, Parag P Shah, Mark A Doll, et al.
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