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Mark B Pepys

Showing results (41-50 of 82) with videos related to

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Cell Reports|December 3, 2015
Multinucleated Giant Cells Are Specialized for Complement-Mediated Phagocytosis and Large Target DestructionRonny Milde, Julia Ritter, Glenys A Tennent, et al.
Blood|October 8, 2005
Sequential heart and autologous stem cell transplantation for systemic AL amyloidosisJulian D Gillmore, Hugh J Goodman, Helen J Lachmann, et al.
Lancet (London, England)|February 14, 2003
MHC typing in variant Creutzfeldt-Jakob diseaseMark B Pepys, Alison Bybee, David R Booth, et al.
Open Biology|February 5, 2016
Pharmacological removal of serum amyloid P component from intracerebral plaques and cerebrovascular Aβ amyloid deposits in vivoRaya Al-Shawi, Glenys A Tennent, David J Millar, et al.
Circulation|June 9, 2004
Obesity is an important determinant of baseline serum C-reactive protein concentration in monozygotic twins, independent of genetic influencesJerry R Greenfield, Katherine Samaras, Arthur B Jenkins, et al.
The New England Journal of Medicine|April 9, 2004
C-reactive protein and other circulating markers of inflammation in the prediction of coronary heart diseaseJohn Danesh, Jeremy G Wheeler, Gideon M Hirschfield, et al.
The New England Journal of Medicine|June 7, 2002
Misdiagnosis of hereditary amyloidosis as AL (primary) amyloidosisHelen J Lachmann, David R Booth, Susanne E Booth, et al.
Circulation Research|October 29, 2005
Proinflammatory effects of bacterial recombinant human C-reactive protein are caused by contamination with bacterial products, not by C-reactive protein itselfMark B Pepys, Philip N Hawkins, Melvyn C Kahan, et al.
The Journal of Infectious Diseases|March 18, 2006
Human serum amyloid P component protects against Escherichia coli O157:H7 Shiga toxin 2 in vivo: therapeutic implications for hemolytic-uremic syndromeGlen D Armstrong, George L Mulvey, Paola Marcato, et al.
Atherosclerosis|June 26, 2007
Transgenic human CRP is not pro-atherogenic, pro-atherothrombotic or pro-inflammatory in apoE-/- miceGlenys A Tennent, Winston L Hutchinson, Melvyn C Kahan, et al.
Pageof 9

Showing results (41-50 of 82) with videos related to

Sort By:
Pageof 9
Cell Reports|December 3, 2015
Multinucleated Giant Cells Are Specialized for Complement-Mediated Phagocytosis and Large Target DestructionRonny Milde, Julia Ritter, Glenys A Tennent, et al.
Blood|October 8, 2005
Sequential heart and autologous stem cell transplantation for systemic AL amyloidosisJulian D Gillmore, Hugh J Goodman, Helen J Lachmann, et al.
Lancet (London, England)|February 14, 2003
MHC typing in variant Creutzfeldt-Jakob diseaseMark B Pepys, Alison Bybee, David R Booth, et al.
Open Biology|February 5, 2016
Pharmacological removal of serum amyloid P component from intracerebral plaques and cerebrovascular Aβ amyloid deposits in vivoRaya Al-Shawi, Glenys A Tennent, David J Millar, et al.
Circulation|June 9, 2004
Obesity is an important determinant of baseline serum C-reactive protein concentration in monozygotic twins, independent of genetic influencesJerry R Greenfield, Katherine Samaras, Arthur B Jenkins, et al.
The New England Journal of Medicine|April 9, 2004
C-reactive protein and other circulating markers of inflammation in the prediction of coronary heart diseaseJohn Danesh, Jeremy G Wheeler, Gideon M Hirschfield, et al.
The New England Journal of Medicine|June 7, 2002
Misdiagnosis of hereditary amyloidosis as AL (primary) amyloidosisHelen J Lachmann, David R Booth, Susanne E Booth, et al.
Circulation Research|October 29, 2005
Proinflammatory effects of bacterial recombinant human C-reactive protein are caused by contamination with bacterial products, not by C-reactive protein itselfMark B Pepys, Philip N Hawkins, Melvyn C Kahan, et al.
The Journal of Infectious Diseases|March 18, 2006
Human serum amyloid P component protects against Escherichia coli O157:H7 Shiga toxin 2 in vivo: therapeutic implications for hemolytic-uremic syndromeGlen D Armstrong, George L Mulvey, Paola Marcato, et al.
Atherosclerosis|June 26, 2007
Transgenic human CRP is not pro-atherogenic, pro-atherothrombotic or pro-inflammatory in apoE-/- miceGlenys A Tennent, Winston L Hutchinson, Melvyn C Kahan, et al.
Pageof 9