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Proceedings of the National Academy of Sciences of the United States of America|July 9, 2015
Inhibition of MCU forces extramitochondrial adaptations governing physiological and pathological stress responses in heartTyler P Rasmussen, Yuejin Wu, Mei-ling A Joiner, et al.Nature Communications|May 27, 2021
CaMKII oxidation is a critical performance/disease trade-off acquired at the dawn of vertebrate evolutionQinchuan Wang, Erick O Hernández-Ochoa, Meera C Viswanathan, et al.The Journal of Clinical Investigation|August 5, 2020
MICAL1 constrains cardiac stress responses and protects against disease by oxidizing CaMKIIKlitos Konstantinidis, Vassilios J Bezzerides, Lo Lai, et al.Science Translational Medicine|July 26, 2013
CaMKII is essential for the proasthmatic effects of oxidationPhilip N Sanders, Olha M Koval, Omar A Jaffer, et al.Pageof 19