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Journal of Molecular and Cellular Cardiology|February 14, 2012
MyD88 mediated inflammatory signaling leads to CaMKII oxidation, cardiac hypertrophy and death after myocardial infarctionMadhu V Singh, Paari D Swaminathan, Elizabeth D Luczak, et al.
The Journal of Asthma : Official Journal of the Association for the Care of Asthma|July 30, 2004
Successful school-based intervention for inner-city children with persistent asthmaMark E Anderson, Michelle R Freas, Andrea S Wallace, et al.
Channels (Austin, Tex.)|August 12, 2008
Calmodulin kinase II inhibition enhances ischemic preconditioning by augmenting ATP-sensitive K+ currentJingdong Li, Celine Marionneau, Olha Koval, et al.
Nature Structural & Molecular Biology|February 26, 2004
An EF-hand in the sodium channel couples intracellular calcium to cardiac excitabilityTammy L Wingo, Vikas N Shah, Mark E Anderson, et al.
Plos One|August 6, 2014
Inhibition of CaMKII does not attenuate cardiac hypertrophy in mice with dysfunctional ryanodine receptorAsima Chakraborty, Daniel A Pasek, Tai-Qin Huang, et al.
Circulation|February 13, 2002
Calmodulin inhibitor W-7 unmasks a novel electrocardiographic parameter that predicts initiation of torsade de pointesT David Gbadebo, Robert W Trimble, Michelle S C Khoo, et al.
The Journal of Biological Chemistry|October 11, 2022
Oxidative stress-induced autonomous activation of the calcium/calmodulin-dependent kinase II involves disulfide formation in the regulatory domainNathália Rocco-Machado, Lo Lai, Geumsoo Kim, et al.
Lab on a Chip|July 23, 2004
A microfluidic device to confine a single cardiac myocyte in a sub-nanoliter volume on planar microelectrodes for extracellular potential recordingsAndreas A Werdich, Eduardo A Lima, Borislav Ivanov, et al.
Cancer Research|January 23, 2013
G-protein inactivator RGS6 mediates myocardial cell apoptosis and cardiomyopathy caused by doxorubicinJianqi Yang, Biswanath Maity, Jie Huang, et al.
American Journal of Physiology. Heart and Circulatory Physiology|March 25, 2008
Differential effects of phospholamban and Ca2+/calmodulin-dependent kinase II on [Ca2+]i transients in cardiac myocytes at physiological stimulation frequenciesAndreas A Werdich, Eduardo A Lima, Igor Dzhura, et al.
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