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Kidney International|May 20, 2004
Proteinuria, a target for renoprotection in patients with type 2 diabetic nephropathy: lessons from RENAALDick de Zeeuw, Giuseppe Remuzzi, Hans-Henrik Parving, et al.Hypertension (Dallas, Tex. : 1979)|April 7, 2010
RILLKKMPSV influences the vasculature, neurons and glia, and (pro)renin receptor expression in the retinaJennifer L Wilkinson-Berka, Ronen Heine, Genevieve Tan, et al.Circulation|July 10, 2002
Prevention of accelerated atherosclerosis by angiotensin-converting enzyme inhibition in diabetic apolipoprotein E-deficient miceRiccardo Candido, Karin A Jandeleit-Dahm, Zemin Cao, et al.Journal of the American Society of Nephrology : JASN|November 23, 2013
Nephropathy and elevated BP in mice with podocyte-specific NADPH oxidase 5 expressionChet E Holterman, Jean-François Thibodeau, Chelsea Towaij, et al.Journal of Clinical Medicine|January 8, 2023
Whether Renal Pathology Is an Independent Predictor for End-Stage Renal Disease in Diabetic Kidney Disease Patients with Nephrotic Range Proteinuria: A Biopsy-Based StudyTingli Wang, Junlin Zhang, Yiting Wang, et al.Diabetes, Obesity & Metabolism|May 4, 2019
Response of 1,5-anhydroglucitol level to intensive glucose- and blood-pressure lowering interventions, and its associations with clinical outcomes in the ADVANCE trialElizabeth Selvin, Dan Wang, John William McEvoy, et al.Diabetes|October 19, 2010
miR-200a Prevents renal fibrogenesis through repression of TGF-β2 expressionBo Wang, Philip Koh, Catherine Winbanks, et al.Diabetes Care|August 9, 2015
Relationship between levels of advanced glycation end products and their soluble receptor and adverse outcomes in adults with type 2 diabetesMerlin C Thomas, Mark Woodward, Bruce Neal, et al.Nephrology (Carlton, Vic.)|October 11, 2012
Targeting the AGE-RAGE axis improves renal function in the context of a healthy diet low in advanced glycation end-product contentVicki Thallas-Bonke, Melinda T Coughlan, Adeline Ly Tan, et al.Diabetes|April 16, 2010
E-cadherin expression is regulated by miR-192/215 by a mechanism that is independent of the profibrotic effects of transforming growth factor-betaBo Wang, Michal Herman-Edelstein, Philip Koh, et al.Pageof 54