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Brain Research|July 16, 2013
Differential effect of D623N variant and wild-type Na(v)1.7 sodium channels on resting potential and interspike membrane potential of dorsal root ganglion neuronsHye-Sook Ahn, Dmytro V Vasylyev, Mark Estacion, et al.
Molecular Pharmacology|August 24, 2018
The Novel Activity of Carbamazepine as an Activation Modulator Extends from NaV1.7 Mutations to the NaV1.8-S242T Mutant Channel from a Patient with Painful Diabetic NeuropathyChongyang Han, Andreas C Themistocleous, Mark Estacion, et al.
Frontiers in Pharmacology|January 7, 2022
Lacosamide Inhibition of NaV1.7 Channels Depends on its Interaction With the Voltage Sensor Domain and the Channel PoreJulie I R Labau, Matthew Alsaloum, Mark Estacion, et al.
Pain|December 10, 2019
Dexpramipexole blocks Nav1.8 sodium channels and provides analgesia in multiple nociceptive and neuropathic pain modelsMatteo Urru, Mirko Muzzi, Elisabetta Coppi, et al.
Brain Communications|September 24, 2021
KCNQ variants and pain modulation: a missense variant in Kv7.3 contributes to pain resilienceJun-Hui Yuan, Mark Estacion, Malgorzata A Mis, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|May 19, 2022
Depolarizing NaV and Hyperpolarizing KV Channels Are Co-Trafficked in Sensory NeuronsGrant P Higerd-Rusli, Matthew Alsaloum, Sidharth Tyagi, et al.
Molecular Pain|May 2, 2019
A gain-of-function sodium channel β2-subunit mutation in painful diabetic neuropathyMatthew Alsaloum, Mark Estacion, Rowida Almomani, et al.
Pain|December 16, 2020
Two independent mouse lines carrying the Nav1.7 I228M gain-of-function variant display dorsal root ganglion neuron hyperexcitability but a minimal pain phenotypeLubin Chen, Nivanthika K Wimalasena, Jaehoon Shim, et al.
The Lancet. Neurology|February 21, 2017
Safety and efficacy of a Nav1.7 selective sodium channel blocker in patients with trigeminal neuralgia: a double-blind, placebo-controlled, randomised withdrawal phase 2a trialJoanna M Zakrzewska, Joanne Palmer, Valerie Morisset, et al.
Brain : a Journal of Neurology|April 30, 2014
Gain-of-function mutations in sodium channel Na(v)1.9 in painful neuropathyJianying Huang, Chongyang Han, Mark Estacion, et al.
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