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The Journal of Biological Chemistry|September 19, 2006
The inhibition of mitochondrial complex I (NADH:ubiquinone oxidoreductase) by Zn2+Mark S Sharpley, Judy HirstBiochemistry|February 18, 2009
Reduction of hydrophilic ubiquinones by the flavin in mitochondrial NADH:ubiquinone oxidoreductase (Complex I) and production of reactive oxygen speciesMartin S King, Mark S Sharpley, Judy HirstBiochemistry|January 4, 2006
Interactions between phospholipids and NADH:ubiquinone oxidoreductase (complex I) from bovine mitochondriaMark S Sharpley, Richard J Shannon, Federica Draghi, et al.Developmental Cell|August 24, 2021
Metabolic plasticity drives development during mammalian embryogenesisMark S Sharpley, Fangtao Chi, Johanna Ten Hoeve, et al.Developmental Cell|March 21, 2020
Glycolysis-Independent Glucose Metabolism Distinguishes TE from ICM Fate during Mammalian EmbryogenesisFangtao Chi, Mark S Sharpley, Raghavendra Nagaraj, et al.Cell|January 14, 2017
Nuclear Localization of Mitochondrial TCA Cycle Enzymes as a Critical Step in Mammalian Zygotic Genome ActivationRaghavendra Nagaraj, Mark S Sharpley, Fangtao Chi, et al.Elife|October 29, 2021
Paths and pathways that generate cell-type heterogeneity and developmental progression in hematopoiesisJuliet R Girard, Lauren M Goins, Dung M Vuu, et al.The Journal of Biological Chemistry|March 21, 2007
Interaction of the mitochondria-targeted antioxidant MitoQ with phospholipid bilayers and ubiquinone oxidoreductasesAndrew M James, Mark S Sharpley, Abdul-Rahman B Manas, et al.Cell|October 16, 2012
Heteroplasmy of mouse mtDNA is genetically unstable and results in altered behavior and cognitionMark S Sharpley, Christine Marciniak, Kristin Eckel-Mahan, et al.Proceedings of the National Academy of Sciences of the United States of America|November 7, 2012
Mouse mtDNA mutant model of Leber hereditary optic neuropathyChun Shi Lin, Mark S Sharpley, Weiwei Fan, et al.Pageof 2