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Autophagy|July 31, 2013
MTORC1 determines autophagy through ULK1 regulation in skeletal musclePerrine Castets, Markus A RüeggBioarchitecture|April 12, 2014
In vivo evidence for mTORC2-mediated actin cytoskeleton rearrangement in neuronsNico Angliker, Markus A RüeggCell Research|March 15, 2017
LncRNA-encoded peptides: More than translational noise?Nathalie Rion, Markus A RüeggAnnual Review of Pharmacology and Toxicology|October 13, 2010
Molecular mechanisms and treatment options for muscle wasting diseasesMarkus A Rüegg, David J GlassFrontiers in Molecular Neuroscience|September 28, 2020
The TOR Pathway at the Neuromuscular Junction: More Than a Metabolic Player?Perrine Castets, Daniel J Ham, Markus A RüeggPhysiological Reviews|June 26, 2015
Mechanisms Regulating Neuromuscular Junction Development and Function and Causes of Muscle WastingLionel A Tintignac, Hans-Rudolf Brenner, Markus A RüeggJournal of Neuromuscular Diseases|November 18, 2016
"Get the Balance Right": Pathological Significance of Autophagy Perturbation in Neuromuscular DisordersPerrine Castets, Stephan Frank, Michael Sinnreich, et al.Journal of Neuromuscular Diseases|November 19, 2016
Best Practices and Standard Protocols as a Tool to Enhance Translation for Neuromuscular DisordersRaffaella Willmann, Annamaria De Luca, Kanneboyina Nagaraju, et al.Plos One|September 7, 2012
Fatigue and muscle atrophy in a mouse model of myasthenia gravis is paralleled by loss of sarcolemmal nNOSSarina Meinen, Shuo Lin, Markus A Rüegg, et al.Matrix Biology : Journal of the International Society for Matrix Biology|March 19, 2026
Laminin-α2 is required for the maintenance of the myotendinous junction in vivoJulia Schedel, Shuo Lin, Thomas Bock, et al.Pageof 10