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The Journal of Clinical Investigation|May 25, 2011
mTORC1 activation in podocytes is a critical step in the development of diabetic nephropathy in miceKen Inoki, Hiroyuki Mori, Junying Wang, et al.Nature Communications|September 16, 2025
Neural xenografts contribute to long-term recovery in stroke via molecular graft-host crosstalkRebecca Z Weber, Beatriz Achón Buil, Nora H Rentsch, et al.American Journal of Human Genetics|July 28, 2009
Identification of an agrin mutation that causes congenital myasthenia and affects synapse functionCaroline Huzé, Stéphanie Bauché, Pascale Richard, et al.The Journal of Clinical Investigation|May 25, 2011
Role of mTOR in podocyte function and diabetic nephropathy in humans and miceMarkus Gödel, Björn Hartleben, Nadja Herbach, et al.The Journal of Cell Biology|December 17, 2009
Muscle inactivation of mTOR causes metabolic and dystrophin defects leading to severe myopathyValérie Risson, Laetitia Mazelin, Mila Roceri, et al.Pageof 10