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Markus Christmann

Showing results (21-30 of 72) with videos related to

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Carcinogenesis|July 11, 2006
A role for UV-light-induced c-Fos: Stimulation of nucleotide excision repair and protection against sustained JNK activation and apoptosisMarkus Christmann, Maja T Tomicic, Dorthe Aasland, et al.
Toxicology and Applied Pharmacology|July 16, 2005
Apaf-1 deficient mouse fibroblasts are resistant to MNNG and MMS-induced apoptotic death without attenuation of Bcl-2 declineMaja T Tomicic, Markus Christmann, Kerstin Fabian, et al.
DNA Repair|May 9, 2007
MGMT: key node in the battle against genotoxicity, carcinogenicity and apoptosis induced by alkylating agentsBernd Kaina, Markus Christmann, Steffen Naumann, et al.
Toxicology|November 6, 2003
Mechanisms of human DNA repair: an updateMarkus Christmann, Maja T Tomicic, Wynand P Roos, et al.
Cardiovascular Research|September 23, 2003
Hypertrophic agonists induce the binding of c-Fos to an AP-1 site in cardiac myocytes: implications for the expression of GLUT1Tomàs Santalucía, Markus Christmann, Magdi H Yacoub, et al.
Biomedicine & Pharmacotherapy = Biomedecine & Pharmacotherapie|November 3, 2024
The cell cycle inhibitor p21<sup>CIP1</sup> is essential for irinotecan-induced senescence and plays a decisive role in re-sensitization of temozolomide-resistant glioblastoma cells to irinotecanJason Sallbach, Melanie Woods, Birgit Rasenberger, et al.
Cancers|April 30, 2021
Oxaliplatin-Induced Senescence in Colorectal Cancer Cells Depends on p14<sup>ARF</sup>-Mediated Sustained p53 ActivationMaja T Tomicic, Franziska Krämer, Alexandra Nguyen, et al.
Biochimica Et Biophysica Acta|April 13, 2013
Human three prime exonuclease TREX1 is induced by genotoxic stress and involved in protection of glioma and melanoma cells to anticancer drugsMaja T Tomicic, Dorthe Aasland, Teodora Nikolova, et al.
Nucleic Acids Research|November 30, 2006
c-Fos is required for excision repair of UV-light induced DNA lesions by triggering the re-synthesis of XPFMarkus Christmann, Maja T Tomicic, Judith Origer, et al.
Nucleic Acids Research|June 1, 2010
Three prime exonuclease I (TREX1) is Fos/AP-1 regulated by genotoxic stress and protects against ultraviolet light and benzo(a)pyrene-induced DNA damageMarkus Christmann, Maja T Tomicic, Dorthe Aasland, et al.
Pageof 8

Showing results (21-30 of 72) with videos related to

Sort By:
Pageof 8
Carcinogenesis|July 11, 2006
A role for UV-light-induced c-Fos: Stimulation of nucleotide excision repair and protection against sustained JNK activation and apoptosisMarkus Christmann, Maja T Tomicic, Dorthe Aasland, et al.
Toxicology and Applied Pharmacology|July 16, 2005
Apaf-1 deficient mouse fibroblasts are resistant to MNNG and MMS-induced apoptotic death without attenuation of Bcl-2 declineMaja T Tomicic, Markus Christmann, Kerstin Fabian, et al.
DNA Repair|May 9, 2007
MGMT: key node in the battle against genotoxicity, carcinogenicity and apoptosis induced by alkylating agentsBernd Kaina, Markus Christmann, Steffen Naumann, et al.
Toxicology|November 6, 2003
Mechanisms of human DNA repair: an updateMarkus Christmann, Maja T Tomicic, Wynand P Roos, et al.
Cardiovascular Research|September 23, 2003
Hypertrophic agonists induce the binding of c-Fos to an AP-1 site in cardiac myocytes: implications for the expression of GLUT1Tomàs Santalucía, Markus Christmann, Magdi H Yacoub, et al.
Biomedicine & Pharmacotherapy = Biomedecine & Pharmacotherapie|November 3, 2024
The cell cycle inhibitor p21<sup>CIP1</sup> is essential for irinotecan-induced senescence and plays a decisive role in re-sensitization of temozolomide-resistant glioblastoma cells to irinotecanJason Sallbach, Melanie Woods, Birgit Rasenberger, et al.
Cancers|April 30, 2021
Oxaliplatin-Induced Senescence in Colorectal Cancer Cells Depends on p14<sup>ARF</sup>-Mediated Sustained p53 ActivationMaja T Tomicic, Franziska Krämer, Alexandra Nguyen, et al.
Biochimica Et Biophysica Acta|April 13, 2013
Human three prime exonuclease TREX1 is induced by genotoxic stress and involved in protection of glioma and melanoma cells to anticancer drugsMaja T Tomicic, Dorthe Aasland, Teodora Nikolova, et al.
Nucleic Acids Research|November 30, 2006
c-Fos is required for excision repair of UV-light induced DNA lesions by triggering the re-synthesis of XPFMarkus Christmann, Maja T Tomicic, Judith Origer, et al.
Nucleic Acids Research|June 1, 2010
Three prime exonuclease I (TREX1) is Fos/AP-1 regulated by genotoxic stress and protects against ultraviolet light and benzo(a)pyrene-induced DNA damageMarkus Christmann, Maja T Tomicic, Dorthe Aasland, et al.
Pageof 8