Jove
Visualize
Contact Us
JoVE
x logofacebook logolinkedin logoyoutube logo
ABOUT JoVE
OverviewLeadershipBlogJoVE Help Center
AUTHORS
Publishing ProcessEditorial BoardScope & PoliciesPeer ReviewFAQSubmit
LIBRARIANS
TestimonialsSubscriptionsAccessResourcesLibrary Advisory BoardFAQ
RESEARCH
JoVE JournalMethods CollectionsJoVE Encyclopedia of ExperimentsArchive
EDUCATION
JoVE CoreJoVE BusinessJoVE Science EducationJoVE Lab ManualFaculty Resource CenterFaculty Site
Terms & Conditions of Use
Privacy Policy
Policies

Filters

Markus Christmann

Showing results (31-40 of 72) with videos related to

Pageof 8
Sort By:
Archives of Toxicology|July 11, 2019
Functional mismatch repair and inactive p53 drive sensitization of colorectal cancer cells to irinotecan via the IAP antagonist BV6Maja T Tomicic, Christian Steigerwald, Birgit Rasenberger, et al.
Proceedings of the National Academy of Sciences of the United States of America|December 14, 2011
Human monocytes are severely impaired in base and DNA double-strand break repair that renders them vulnerable to oxidative stressMartina Bauer, Michael Goldstein, Markus Christmann, et al.
Molecular Pharmacology|January 16, 2003
Apoptosis induced by (E)-5-(2-bromovinyl)-2'-deoxyuridine in varicella zoster virus thymidine kinase-expressing cells is driven by activation of c-Jun/activator protein-1 and Fas ligand/caspase-8Maja T Tomicic, Claudia Friedrichs, Markus Christmann, et al.
Cellular and Molecular Life Sciences : CMLS|October 27, 2010
Delayed c-Fos activation in human cells triggers XPF induction and an adaptive response to UVC-induced DNA damage and cytotoxicityMaja T Tomicic, Patricia Reischmann, Birgit Rasenberger, et al.
Clinical Epigenetics|May 10, 2016
MGMT promoter methylation determined by HRM in comparison to MSP and pyrosequencing for predicting high-grade glioma responseOlivier J Switzeny, Markus Christmann, Mirjam Renovanz, et al.
International Journal of Cancer|February 5, 2010
MGMT activity, promoter methylation and immunohistochemistry of pretreatment and recurrent malignant gliomas: a comparative study on astrocytoma and glioblastomaMarkus Christmann, Georg Nagel, Sigrid Horn, et al.
International Journal of Cancer|September 24, 2004
Long-term activation of SAPK/JNK, p38 kinase and fas-L expression by cisplatin is attenuated in human carcinoma cells that acquired drug resistanceAnamaria Brozovic, Gerhard Fritz, Markus Christmann, et al.
DNA Repair|September 23, 2008
WRN protects against topo I but not topo II inhibitors by preventing DNA break formationMarkus Christmann, Maja T Tomicic, Christopher Gestrich, et al.
Cancer Research|December 20, 2007
Differential sensitivity of malignant glioma cells to methylating and chloroethylating anticancer drugs: p53 determines the switch by regulating xpc, ddb2, and DNA double-strand breaksLuís F Z Batista, Wynand P Roos, Markus Christmann, et al.
Journal of Neurochemistry|November 23, 2017
Repair gene O<sup>6</sup> -methylguanine-DNA methyltransferase is controlled by SP1 and up-regulated by glucocorticoids, but not by temozolomide and radiationDorthe Aasland, Thomas R Reich, Maja T Tomicic, et al.
Pageof 8

Showing results (31-40 of 72) with videos related to

Sort By:
Pageof 8
Archives of Toxicology|July 11, 2019
Functional mismatch repair and inactive p53 drive sensitization of colorectal cancer cells to irinotecan via the IAP antagonist BV6Maja T Tomicic, Christian Steigerwald, Birgit Rasenberger, et al.
Proceedings of the National Academy of Sciences of the United States of America|December 14, 2011
Human monocytes are severely impaired in base and DNA double-strand break repair that renders them vulnerable to oxidative stressMartina Bauer, Michael Goldstein, Markus Christmann, et al.
Molecular Pharmacology|January 16, 2003
Apoptosis induced by (E)-5-(2-bromovinyl)-2'-deoxyuridine in varicella zoster virus thymidine kinase-expressing cells is driven by activation of c-Jun/activator protein-1 and Fas ligand/caspase-8Maja T Tomicic, Claudia Friedrichs, Markus Christmann, et al.
Cellular and Molecular Life Sciences : CMLS|October 27, 2010
Delayed c-Fos activation in human cells triggers XPF induction and an adaptive response to UVC-induced DNA damage and cytotoxicityMaja T Tomicic, Patricia Reischmann, Birgit Rasenberger, et al.
Clinical Epigenetics|May 10, 2016
MGMT promoter methylation determined by HRM in comparison to MSP and pyrosequencing for predicting high-grade glioma responseOlivier J Switzeny, Markus Christmann, Mirjam Renovanz, et al.
International Journal of Cancer|February 5, 2010
MGMT activity, promoter methylation and immunohistochemistry of pretreatment and recurrent malignant gliomas: a comparative study on astrocytoma and glioblastomaMarkus Christmann, Georg Nagel, Sigrid Horn, et al.
International Journal of Cancer|September 24, 2004
Long-term activation of SAPK/JNK, p38 kinase and fas-L expression by cisplatin is attenuated in human carcinoma cells that acquired drug resistanceAnamaria Brozovic, Gerhard Fritz, Markus Christmann, et al.
DNA Repair|September 23, 2008
WRN protects against topo I but not topo II inhibitors by preventing DNA break formationMarkus Christmann, Maja T Tomicic, Christopher Gestrich, et al.
Cancer Research|December 20, 2007
Differential sensitivity of malignant glioma cells to methylating and chloroethylating anticancer drugs: p53 determines the switch by regulating xpc, ddb2, and DNA double-strand breaksLuís F Z Batista, Wynand P Roos, Markus Christmann, et al.
Journal of Neurochemistry|November 23, 2017
Repair gene O<sup>6</sup> -methylguanine-DNA methyltransferase is controlled by SP1 and up-regulated by glucocorticoids, but not by temozolomide and radiationDorthe Aasland, Thomas R Reich, Maja T Tomicic, et al.
Pageof 8