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Markus Christmann

Showing results (51-60 of 72) with videos related to

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Oncotarget|January 6, 2015
B-Raf inhibitor vemurafenib in combination with temozolomide and fotemustine in the killing response of malignant melanoma cellsWynand P Roos, Steve Quiros, Andrea Krumm, et al.
Nucleic Acids Research|October 4, 2016
Adaptive upregulation of DNA repair genes following benzo(a)pyrene diol epoxide protects against cell death at the expense of mutationsMarkus Christmann, Catherine Boisseau, Rebekka Kitzinger, et al.
Archives of Toxicology|May 21, 2026
Points of Departure for BPDE-induced cellular senescence and cell death are characterized by altered DNA damage signaling and regulated by unrepaired DNA double-strand breaksAriane Schmidt, Anna Schöneis, Jason Sallbach, et al.
Cells|August 2, 2019
Lipoic Acid Synergizes with Antineoplastic Drugs in Colorectal Cancer by Targeting p53 for Proteasomal DegradationCarina Neitzel, Nina Seiwert, Anja Göder, et al.
Cancers|May 14, 2022
Senescence Is the Main Trait Induced by Temozolomide in Glioblastoma CellsLea Beltzig, Christian Schwarzenbach, Petra Leukel, et al.
Molecular Cancer Therapeutics|November 9, 2021
The Mitochondrial Disruptor Devimistat (CPI-613) Synergizes with Genotoxic Anticancer Drugs in Colorectal Cancer Therapy in a Bim-Dependent MannerCarina Arnold, Philipp Demuth, Nina Seiwert, et al.
Nature Communications|February 24, 2018
HDAC1 and HDAC2 integrate checkpoint kinase phosphorylation and cell fate through the phosphatase-2A subunit PR130Anja Göder, Claudia Emmerich, Teodora Nikolova, et al.
Oncotarget|August 4, 2016
Integrin αVβ3 silencing sensitizes malignant glioma cells to temozolomide by suppression of homologous recombination repairMarkus Christmann, Kathrin Diesler, Dragomira Majhen, et al.
Cell Death & Disease|May 3, 2025
Therapy-induced senescence of glioblastoma cells is determined by the p21<sup>CIP1</sup>-CDK1/2 axis and does not require activation of DREAMChristian Schwarzenbach, Justus Rinke, Juliana B Vilar, et al.
Psychoneuroendocrinology|June 1, 2026
The association between telomere length and expression of DNA repair genes as a biological trace of cumulative lifetime stressHannah Keppler, R Nehir Mavioglu, Alexander Behnke, et al.
Pageof 8

Showing results (51-60 of 72) with videos related to

Sort By:
Pageof 8
Oncotarget|January 6, 2015
B-Raf inhibitor vemurafenib in combination with temozolomide and fotemustine in the killing response of malignant melanoma cellsWynand P Roos, Steve Quiros, Andrea Krumm, et al.
Nucleic Acids Research|October 4, 2016
Adaptive upregulation of DNA repair genes following benzo(a)pyrene diol epoxide protects against cell death at the expense of mutationsMarkus Christmann, Catherine Boisseau, Rebekka Kitzinger, et al.
Archives of Toxicology|May 21, 2026
Points of Departure for BPDE-induced cellular senescence and cell death are characterized by altered DNA damage signaling and regulated by unrepaired DNA double-strand breaksAriane Schmidt, Anna Schöneis, Jason Sallbach, et al.
Cells|August 2, 2019
Lipoic Acid Synergizes with Antineoplastic Drugs in Colorectal Cancer by Targeting p53 for Proteasomal DegradationCarina Neitzel, Nina Seiwert, Anja Göder, et al.
Cancers|May 14, 2022
Senescence Is the Main Trait Induced by Temozolomide in Glioblastoma CellsLea Beltzig, Christian Schwarzenbach, Petra Leukel, et al.
Molecular Cancer Therapeutics|November 9, 2021
The Mitochondrial Disruptor Devimistat (CPI-613) Synergizes with Genotoxic Anticancer Drugs in Colorectal Cancer Therapy in a Bim-Dependent MannerCarina Arnold, Philipp Demuth, Nina Seiwert, et al.
Nature Communications|February 24, 2018
HDAC1 and HDAC2 integrate checkpoint kinase phosphorylation and cell fate through the phosphatase-2A subunit PR130Anja Göder, Claudia Emmerich, Teodora Nikolova, et al.
Oncotarget|August 4, 2016
Integrin αVβ3 silencing sensitizes malignant glioma cells to temozolomide by suppression of homologous recombination repairMarkus Christmann, Kathrin Diesler, Dragomira Majhen, et al.
Cell Death & Disease|May 3, 2025
Therapy-induced senescence of glioblastoma cells is determined by the p21<sup>CIP1</sup>-CDK1/2 axis and does not require activation of DREAMChristian Schwarzenbach, Justus Rinke, Juliana B Vilar, et al.
Psychoneuroendocrinology|June 1, 2026
The association between telomere length and expression of DNA repair genes as a biological trace of cumulative lifetime stressHannah Keppler, R Nehir Mavioglu, Alexander Behnke, et al.
Pageof 8