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Microorganisms|March 29, 2020
Cysteine Residues in <i>Helicobacter pylori</i> Adhesin HopQ are Required for CEACAM-HopQ Interaction and Subsequent CagA TranslocationYoussef Hamway, Karin Taxauer, Kristof Moonens, et al.
Plos One|February 21, 2012
Evidence for conserved function of γ-glutamyltranspeptidase in Helicobacter genusMirko Rossi, Christian Bolz, Joana Revez, et al.
Cellular and Molecular Gastroenterology and Hepatology|November 14, 2020
Loss of RNF43 Function Contributes to Gastric Carcinogenesis by Impairing DNA Damage ResponseVictoria Neumeyer, Anna Brutau-Abia, Michael Allgäuer, et al.
Chemistry (Weinheim an Der Bergstrasse, Germany)|May 28, 2011
Synthesis of 5-acetyloxazoles and 1,2-diketones from β-alkoxy-β-ketoenamides and their subsequent transformationsTilman Lechel, Markus Gerhard, Daniel Trawny, et al.
Gastroenterology|May 12, 2005
A secreted low-molecular-weight protein from Helicobacter pylori induces cell-cycle arrest of T cellsMarkus Gerhard, Christian Schmees, Petra Voland, et al.
Infection and Immunity|January 27, 2004
Functional analysis of the cag pathogenicity island in Helicobacter pylori isolates from patients with gastritis, peptic ulcer, and gastric cancerSteffen Backert, Tobias Schwarz, Stephan Miehlke, et al.
Proteomes|October 4, 2017
Performance of a Multiplex Serological Helicobacter pylori Assay on a Novel Microfluidic Assay PlatformAngela Filomena, Anna Guenther, Hannes Planatscher, et al.
High-Throughput|June 2, 2018
Development of a Bead-Based Multiplex Assay for the Analysis of the Serological Response against the Six Pathogens HAV, HBV, HCV, CMV, T. gondii, and H. pyloriAngela Filomena, Frank Pessler, Manas K Akmatov, et al.
International Journal of Medical Microbiology : IJMM|October 10, 2013
Helicobacter pylori FliD protein is a highly sensitive and specific marker for serologic diagnosis of H. pylori infectionMohammad Khalifeh Gholi, Behnam Kalali, Luca Formichella, et al.
The Journal of Investigative Dermatology|December 17, 2003
Toll-like receptor expression in human keratinocytes: nuclear factor kappaB controlled gene activation by Staphylococcus aureus is toll-like receptor 2 but not toll-like receptor 4 or platelet activating factor receptor dependentMartin Mempel, Verena Voelcker, Gabriele Köllisch, et al.
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