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Markus Kaller

Showing results (11-20 of 32) with videos related to

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Oncotarget|September 7, 2013
Repression of c-Kit by p53 is mediated by miR-34 and is associated with reduced chemoresistance, migration and stemnessHelge Siemens, Rene Jackstadt, Markus Kaller, et al.
Methods in Molecular Biology (Clifton, N.J.)|July 26, 2014
Identification of microRNA targets by pulsed SILACMarkus Kaller, Silke Oeljeklaus, Bettina Warscheid, et al.
Cancers|May 11, 2024
<i>LINC01021</i> Attenuates Expression and Affects Alternative Splicing of a Subset of p53-Regulated GenesMarkus Kaller, Ignasi Forné, Axel Imhof, et al.
Advances in Experimental Medicine and Biology|February 5, 2013
The p53/microRNA network in cancer: experimental and bioinformatics approachesSabine Hünten, Helge Siemens, Markus Kaller, et al.
Cancer Communications (London, England)|January 16, 2024
AP4 induces JNK1 and a miR-22-3p/FOSL1 feed-forward loop to activate AP-1 and promote colorectal cancer metastasisJinjiang Chou, Markus Kaller, Matjaz Rokavec, et al.
Molecular Cancer|May 27, 2022
AP4 suppresses DNA damage, chromosomal instability and senescence via inducing MDC1/Mediator of DNA damage Checkpoint 1 and repressing MIR22HG/miR-22-3pJinjiang Chou, Markus Kaller, Stephanie Jaeckel, et al.
Cellular and Molecular Gastroenterology and Hepatology|April 19, 2020
Characterization of a p53/miR-34a/CSF1R/STAT3 Feedback Loop in Colorectal CancerXiaolong Shi, Markus Kaller, Matjaz Rokavec, et al.
Eukaryotic Cell|March 11, 2008
Dictyostelium Aurora kinase has properties of both Aurora A and Aurora B kinasesHui Li, Qian Chen, Markus Kaller, et al.
International Journal of Biological Sciences|September 23, 2022
<i>Csf1r</i> mediates enhancement of intestinal tumorigenesis caused by inactivation of <i>Mir34a</i>Fangteng Liu, Nassim Bouznad, Markus Kaller, et al.
Cell Cycle (Georgetown, Tex.)|December 3, 2011
miR-34 and SNAIL form a double-negative feedback loop to regulate epithelial-mesenchymal transitionsHelge Siemens, Rene Jackstadt, Sabine Hünten, et al.
Pageof 4

Showing results (11-20 of 32) with videos related to

Sort By:
Pageof 4
Oncotarget|September 7, 2013
Repression of c-Kit by p53 is mediated by miR-34 and is associated with reduced chemoresistance, migration and stemnessHelge Siemens, Rene Jackstadt, Markus Kaller, et al.
Methods in Molecular Biology (Clifton, N.J.)|July 26, 2014
Identification of microRNA targets by pulsed SILACMarkus Kaller, Silke Oeljeklaus, Bettina Warscheid, et al.
Cancers|May 11, 2024
<i>LINC01021</i> Attenuates Expression and Affects Alternative Splicing of a Subset of p53-Regulated GenesMarkus Kaller, Ignasi Forné, Axel Imhof, et al.
Advances in Experimental Medicine and Biology|February 5, 2013
The p53/microRNA network in cancer: experimental and bioinformatics approachesSabine Hünten, Helge Siemens, Markus Kaller, et al.
Cancer Communications (London, England)|January 16, 2024
AP4 induces JNK1 and a miR-22-3p/FOSL1 feed-forward loop to activate AP-1 and promote colorectal cancer metastasisJinjiang Chou, Markus Kaller, Matjaz Rokavec, et al.
Molecular Cancer|May 27, 2022
AP4 suppresses DNA damage, chromosomal instability and senescence via inducing MDC1/Mediator of DNA damage Checkpoint 1 and repressing MIR22HG/miR-22-3pJinjiang Chou, Markus Kaller, Stephanie Jaeckel, et al.
Cellular and Molecular Gastroenterology and Hepatology|April 19, 2020
Characterization of a p53/miR-34a/CSF1R/STAT3 Feedback Loop in Colorectal CancerXiaolong Shi, Markus Kaller, Matjaz Rokavec, et al.
Eukaryotic Cell|March 11, 2008
Dictyostelium Aurora kinase has properties of both Aurora A and Aurora B kinasesHui Li, Qian Chen, Markus Kaller, et al.
International Journal of Biological Sciences|September 23, 2022
<i>Csf1r</i> mediates enhancement of intestinal tumorigenesis caused by inactivation of <i>Mir34a</i>Fangteng Liu, Nassim Bouznad, Markus Kaller, et al.
Cell Cycle (Georgetown, Tex.)|December 3, 2011
miR-34 and SNAIL form a double-negative feedback loop to regulate epithelial-mesenchymal transitionsHelge Siemens, Rene Jackstadt, Sabine Hünten, et al.
Pageof 4