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Markus Moser

Showing results (71-80 of 134) with videos related to

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Cell|July 30, 2008
SILAC mouse for quantitative proteomics uncovers kindlin-3 as an essential factor for red blood cell functionMarcus Krüger, Markus Moser, Siegfried Ussar, et al.
Nature Medicine|February 24, 2009
Leukocyte adhesion deficiency-III is caused by mutations in KINDLIN3 affecting integrin activationLena Svensson, Kimberley Howarth, Alison McDowall, et al.
Leukemia|May 21, 2022
Inability to phosphorylate Y88 of p27<sup>Kip1</sup> enforces reduced p27 protein levels and accelerates leukemia progressionHeidelinde Jäkel, Martin Taschler, Karin Jung, et al.
Human Mutation|April 27, 2004
PKHD1 mutations in families requesting prenatal diagnosis for autosomal recessive polycystic kidney disease (ARPKD)Carsten Bergmann, Jan Senderek, Frank Schneider, et al.
The Journal of Cell Biology|June 27, 2007
The RGD motif in fibronectin is essential for development but dispensable for fibril assemblySeiichiro Takahashi, Michael Leiss, Markus Moser, et al.
Blood|October 7, 2015
Minimal amounts of kindlin-3 suffice for basal platelet and leukocyte functions in miceSarah Klapproth, Federico A Moretti, Marlis Zeiler, et al.
Journal of Bone and Mineral Research : the Official Journal of the American Society for Bone and Mineral Research|September 1, 2012
Inactivation of anoctamin-6/Tmem16f, a regulator of phosphatidylserine scrambling in osteoblasts, leads to decreased mineral deposition in skeletal tissuesHarald W A Ehlen, Milana Chinenkova, Markus Moser, et al.
The Journal of Experimental Medicine|August 19, 2015
Kindlin-3-mediated integrin adhesion is dispensable for quiescent but essential for activated hematopoietic stem cellsRaphael Ruppert, Markus Moser, Markus Sperandio, et al.
Journal of Bone and Mineral Research : the Official Journal of the American Society for Bone and Mineral Research|July 19, 2016
An O-Glycosylation of Fibronectin Mediates Hepatic Osteodystrophy Through α4β1 IntegrinCarla Sens, Eva Altrock, Katrin Rau, et al.
Proceedings of the National Academy of Sciences of the United States of America|September 14, 2021
Selective depletion of a CD64-expressing phagocyte subset mediates protection against toxic kidney injury and failureNatallia Salei, Xingqi Ji, Dalia Pakalniškytė, et al.
Pageof 14

Showing results (71-80 of 134) with videos related to

Sort By:
Pageof 14
Cell|July 30, 2008
SILAC mouse for quantitative proteomics uncovers kindlin-3 as an essential factor for red blood cell functionMarcus Krüger, Markus Moser, Siegfried Ussar, et al.
Nature Medicine|February 24, 2009
Leukocyte adhesion deficiency-III is caused by mutations in KINDLIN3 affecting integrin activationLena Svensson, Kimberley Howarth, Alison McDowall, et al.
Leukemia|May 21, 2022
Inability to phosphorylate Y88 of p27<sup>Kip1</sup> enforces reduced p27 protein levels and accelerates leukemia progressionHeidelinde Jäkel, Martin Taschler, Karin Jung, et al.
Human Mutation|April 27, 2004
PKHD1 mutations in families requesting prenatal diagnosis for autosomal recessive polycystic kidney disease (ARPKD)Carsten Bergmann, Jan Senderek, Frank Schneider, et al.
The Journal of Cell Biology|June 27, 2007
The RGD motif in fibronectin is essential for development but dispensable for fibril assemblySeiichiro Takahashi, Michael Leiss, Markus Moser, et al.
Blood|October 7, 2015
Minimal amounts of kindlin-3 suffice for basal platelet and leukocyte functions in miceSarah Klapproth, Federico A Moretti, Marlis Zeiler, et al.
Journal of Bone and Mineral Research : the Official Journal of the American Society for Bone and Mineral Research|September 1, 2012
Inactivation of anoctamin-6/Tmem16f, a regulator of phosphatidylserine scrambling in osteoblasts, leads to decreased mineral deposition in skeletal tissuesHarald W A Ehlen, Milana Chinenkova, Markus Moser, et al.
The Journal of Experimental Medicine|August 19, 2015
Kindlin-3-mediated integrin adhesion is dispensable for quiescent but essential for activated hematopoietic stem cellsRaphael Ruppert, Markus Moser, Markus Sperandio, et al.
Journal of Bone and Mineral Research : the Official Journal of the American Society for Bone and Mineral Research|July 19, 2016
An O-Glycosylation of Fibronectin Mediates Hepatic Osteodystrophy Through α4β1 IntegrinCarla Sens, Eva Altrock, Katrin Rau, et al.
Proceedings of the National Academy of Sciences of the United States of America|September 14, 2021
Selective depletion of a CD64-expressing phagocyte subset mediates protection against toxic kidney injury and failureNatallia Salei, Xingqi Ji, Dalia Pakalniškytė, et al.
Pageof 14