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The Application of Clinical Genetics|November 8, 2021
Genetic Testing in CYLD Cutaneous Syndrome: An UpdateNikoletta Nagy, Anna Dubois, Marta Szell, et al.Microbes and Infection|May 17, 2005
Microbial compounds induce the expression of pro-inflammatory cytokines, chemokines and human beta-defensin-2 in vaginal epithelial cellsAndor Pivarcsi, Istvan Nagy, Andrea Koreck, et al.Journal of Cellular and Molecular Medicine|August 2, 2008
Molecular response of nasal mucosa to therapeutic exposure to broad-band ultraviolet radiationDavid Mitchell, Lakshmi Paniker, Guillermo Sanchez, et al.Inflammation|October 28, 2010
Polymorphisms of the IL23R gene are associated with psoriasis but not with immunoglobulin A nephropathy in a Hungarian populationEniko Safrany, Marta Szell, Veronika Csongei, et al.Inflammation Research : Official Journal of the European Histamine Research Society ... [Et Al.]|October 25, 2012
Difference of interleukin-23 receptor gene haplotype variants in ulcerative colitis compared to Crohn's disease and psoriasisEniko Safrany, Melinda Szabo, Marta Szell, et al.Clinical Immunology (Orlando, Fla.)|May 26, 2025
Proteomic profiling of serum small extracellular vesicles predicts post-COVID syndrome developmentGabriella Dobra, Edina Gyukity-Sebestyen, Matyas Bukva, et al.World Journal of Gastroenterology|January 19, 2026
Genetic differences in familial adenomatous polyposis syndrome in a Hungarian population: A prospective single center studyTibor Tóth, Renáta Bor, Dóra Nagy, et al.The Journal of Biological Chemistry|April 28, 2005
Identification and characterization of a novel, psoriasis susceptibility-related noncoding RNA gene, PRINSEniko Sonkoly, Zsuzsanna Bata-Csorgo, Andor Pivarcsi, et al.International Journal of Molecular Sciences|January 27, 2024
Missing Heritability in Albinism: Deep Characterization of a Hungarian Albinism Cohort Raises the Possibility of the Digenic Genetic Background of the DiseaseNikoletta Nagy, Margit Pal, Jozsef Kun, et al.The British Journal of Dermatology|March 4, 2026
Targeting non-canonical NF-κB signalling in CYLD cutaneous syndrome by selective inhibition of IκB kinase alphaKirsty Hodgson, Joseph Inns, Gary Reynolds, et al.Pageof 2