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The Journal of General Virology|January 14, 2014
Roles of conserved residues within the pre-NH2-terminal domain of herpes simplex virus 1 DNA polymerase in replication and latency in miceShariya L Terrell, Jean M Pesola, Donald M CoenJournal of Virology|December 28, 2002
The human cytomegalovirus UL97 protein kinase, an antiviral drug target, is required at the stage of nuclear egressPaula M Krosky, Moon-Chang Baek, Donald M CoenMbio|January 17, 2013
Kinetics of facultative heterochromatin and polycomb group protein association with the herpes simplex viral genome during establishment of latent infectionAnna R Cliffe, Donald M Coen, David M KnipeThe Journal of General Virology|May 7, 2010
Association of human cytomegalovirus proteins IRS1 and TRS1 with the viral DNA polymerase accessory subunit UL44Blair L Strang, Adam P Geballe, Donald M CoenMbio|February 10, 2012
Host cell nucleolin is required to maintain the architecture of human cytomegalovirus replication compartmentsBlair L Strang, Steeve Boulant, Tomas Kirchhausen, et al.Journal of Virology|July 13, 2012
Herpes simplex virus is equipped with RNA- and protein-based mechanisms to repress expression of ATRX, an effector of intrinsic immunityIgor Jurak, Leah B Silverstein, Mayuri Sharma, et al.Journal of Virology|August 17, 2004
Specific residues in the connector loop of the human cytomegalovirus DNA polymerase accessory protein UL44 are crucial for interaction with the UL54 catalytic subunitArianna Loregian, Brent A Appleton, James M Hogle, et al.Journal of Virology|November 12, 2005
Herpes simplex virus 1 immediate-early and early gene expression during reactivation from latency under conditions that prevent infectious virus productionJean M Pesola, Jia Zhu, David M Knipe, et al.Journal of Virology|December 13, 2003
Failure of thymidine kinase-negative herpes simplex virus to reactivate from latency following efficient establishmentShih-Heng Chen, Angela Pearson, Donald M Coen, et al.Journal of Virology|June 16, 2006
Low-level expression and reversion both contribute to reactivation of herpes simplex virus drug-resistant mutants with mutations on homopolymeric sequences in thymidine kinaseAnthony Griffiths, Malen A Link, Caroline L Furness, et al.Pageof 13