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The Journal of Biological Chemistry
|
April 17, 2004
Aberrant polycystin-1 expression results in modification of activator protein-1 activity, whereas Wnt signaling remains unaffected
Ngoc Hang Le, Paola van der Bent, Gerwin Huls, et al.
Stroke
|
February 3, 2007
Lacunar infarcts are the main correlate with cognitive dysfunction in CADASIL
Michael K Liem, Jeroen van der Grond, Joost Haan, et al.
Journal of Clinical Immunology
|
March 3, 2016
Deletion of the entire interferon-γ receptor 1 gene causing complete deficiency in three related patients
Inge C de Vor, Pomme M van der Meulen, Vincent Bekker, et al.
British Journal of Haematology
|
July 26, 2002
Minimal residual disease quantification in patients with acute myeloid leukaemia and inv(16)/CBFB-MYH11 gene fusion
Bert A van der Reijden, Annet Simons, Erna Luiten, et al.
European Journal of Medical Genetics
|
June 15, 2010
Additional cryptic CNVs in mentally retarded patients with apparently balanced karyotypes
Antoinet C J Gijsbers, Cathy A J Bosch, Johannes G Dauwerse, et al.
Human Molecular Genetics
|
October 22, 2004
Lowering of Pkd1 expression is sufficient to cause polycystic kidney disease
Irma S Lantinga-van Leeuwen, Johannes G Dauwerse, Hans J Baelde, et al.
American Journal of Human Genetics
|
February 12, 2005
Genetic heterogeneity in Rubinstein-Taybi syndrome: mutations in both the CBP and EP300 genes cause disease
Jeroen H Roelfsema, Stefan J White, Yavuz Ariyürek, et al.
American Journal of Medical Genetics. Part A
|
March 16, 2013
GPSM2 and Chudley-McCullough syndrome: a Dutch founder variant brought to North America
Rowida Almomani, Yu Sun, Emmelien Aten, et al.
European Journal of Endocrinology
|
February 9, 2007
Growth hormone secretion and immunological function of a male patient with a homozygous STAT5b mutation
Marie J E Walenkamp, Solrun Vidarsdottir, Alberto M Pereira, et al.
American Journal of Human Genetics
|
July 6, 2010
Terminal osseous dysplasia is caused by a single recurrent mutation in the FLNA gene
Yu Sun, Rowida Almomani, Emmelien Aten, et al.
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of 12
Search research articles
Search
Showing results (71-80 of 112) with videos related to
Sort By:
Page
of 12
The Journal of Biological Chemistry
|
April 17, 2004
Aberrant polycystin-1 expression results in modification of activator protein-1 activity, whereas Wnt signaling remains unaffected
Ngoc Hang Le, Paola van der Bent, Gerwin Huls, et al.
Stroke
|
February 3, 2007
Lacunar infarcts are the main correlate with cognitive dysfunction in CADASIL
Michael K Liem, Jeroen van der Grond, Joost Haan, et al.
Journal of Clinical Immunology
|
March 3, 2016
Deletion of the entire interferon-γ receptor 1 gene causing complete deficiency in three related patients
Inge C de Vor, Pomme M van der Meulen, Vincent Bekker, et al.
British Journal of Haematology
|
July 26, 2002
Minimal residual disease quantification in patients with acute myeloid leukaemia and inv(16)/CBFB-MYH11 gene fusion
Bert A van der Reijden, Annet Simons, Erna Luiten, et al.
European Journal of Medical Genetics
|
June 15, 2010
Additional cryptic CNVs in mentally retarded patients with apparently balanced karyotypes
Antoinet C J Gijsbers, Cathy A J Bosch, Johannes G Dauwerse, et al.
Human Molecular Genetics
|
October 22, 2004
Lowering of Pkd1 expression is sufficient to cause polycystic kidney disease
Irma S Lantinga-van Leeuwen, Johannes G Dauwerse, Hans J Baelde, et al.
American Journal of Human Genetics
|
February 12, 2005
Genetic heterogeneity in Rubinstein-Taybi syndrome: mutations in both the CBP and EP300 genes cause disease
Jeroen H Roelfsema, Stefan J White, Yavuz Ariyürek, et al.
American Journal of Medical Genetics. Part A
|
March 16, 2013
GPSM2 and Chudley-McCullough syndrome: a Dutch founder variant brought to North America
Rowida Almomani, Yu Sun, Emmelien Aten, et al.
European Journal of Endocrinology
|
February 9, 2007
Growth hormone secretion and immunological function of a male patient with a homozygous STAT5b mutation
Marie J E Walenkamp, Solrun Vidarsdottir, Alberto M Pereira, et al.
American Journal of Human Genetics
|
July 6, 2010
Terminal osseous dysplasia is caused by a single recurrent mutation in the FLNA gene
Yu Sun, Rowida Almomani, Emmelien Aten, et al.
Page
of 12