Jove
Visualize
Contact Us
JoVE
x logofacebook logolinkedin logoyoutube logo
ABOUT JoVE
OverviewLeadershipBlogJoVE Help Center
AUTHORS
Publishing ProcessEditorial BoardScope & PoliciesPeer ReviewFAQSubmit
LIBRARIANS
TestimonialsSubscriptionsAccessResourcesLibrary Advisory BoardFAQ
RESEARCH
JoVE JournalMethods CollectionsJoVE Encyclopedia of ExperimentsArchive
EDUCATION
JoVE CoreJoVE BusinessJoVE Science EducationJoVE Lab ManualFaculty Resource CenterFaculty Site
Terms & Conditions of Use
Privacy Policy
Policies

Filters

Martin Eilers

Showing results (71-80 of 162) with videos related to

Pageof 17
Sort By:
Genes & Development|June 17, 2010
The interaction between Myc and Miz1 is required to antagonize TGFbeta-dependent autocrine signaling during lymphoma formation and maintenanceJan van Riggelen, Judith Müller, Tobias Otto, et al.
Oncogene|January 25, 2003
Myc represses differentiation-induced p21CIP1 expression via Miz-1-dependent interaction with the p21 core promoterSiqin Wu, Cihan Cetinkaya, Maria J Munoz-Alonso, et al.
Cancer Research|December 3, 2010
A SP1/MIZ1/MYCN repression complex recruits HDAC1 at the TRKA and p75NTR promoters and affects neuroblastoma malignancy by inhibiting the cell response to NGFNunzio Iraci, Daniel Diolaiti, Antonella Papa, et al.
The Journal of Clinical Investigation|June 25, 2014
The deubiquitinase USP28 controls intestinal homeostasis and promotes colorectal cancerMarkus E Diefenbacher, Nikita Popov, Sophia M Blake, et al.
Nature Communications|October 4, 2013
Miz1 is required to maintain autophagic fluxElmar Wolf, Anneli Gebhardt, Daisuke Kawauchi, et al.
Cell Cycle (Georgetown, Tex.)|December 4, 2010
TGFβ-dependent gene expression shows that senescence correlates with abortive differentiation along several lineages in Myc-induced lymphomasJudith Müller, Birgit Samans, Jan van Riggelen, et al.
Molecular Cell|February 19, 2011
The MK5/PRAK kinase and Myc form a negative feedback loop that is disrupted during colorectal tumorigenesisTheresia R Kress, Ian G Cannell, Arjan B Brenkman, et al.
Plos One|October 8, 2013
CIP2A influences survival in colon cancer and is critical for maintaining Myc expressionArmin Wiegering, Christina Pfann, Friedrich Wilhelm Uthe, et al.
Nucleic Acids Research|December 19, 2024
Phenotypic screens identify SCAF1 as critical activator of RNAPII elongation and global transcriptionPranjali Bhandare, Ashwin Narain, Julia Hofstetter, et al.
The Journal of Biological Chemistry|June 3, 2010
Sequential activation of NFAT and c-Myc transcription factors mediates the TGF-beta switch from a suppressor to a promoter of cancer cell proliferationGarima Singh, Shiv K Singh, Alexander König, et al.
Pageof 17

Showing results (71-80 of 162) with videos related to

Sort By:
Pageof 17
Genes & Development|June 17, 2010
The interaction between Myc and Miz1 is required to antagonize TGFbeta-dependent autocrine signaling during lymphoma formation and maintenanceJan van Riggelen, Judith Müller, Tobias Otto, et al.
Oncogene|January 25, 2003
Myc represses differentiation-induced p21CIP1 expression via Miz-1-dependent interaction with the p21 core promoterSiqin Wu, Cihan Cetinkaya, Maria J Munoz-Alonso, et al.
Cancer Research|December 3, 2010
A SP1/MIZ1/MYCN repression complex recruits HDAC1 at the TRKA and p75NTR promoters and affects neuroblastoma malignancy by inhibiting the cell response to NGFNunzio Iraci, Daniel Diolaiti, Antonella Papa, et al.
The Journal of Clinical Investigation|June 25, 2014
The deubiquitinase USP28 controls intestinal homeostasis and promotes colorectal cancerMarkus E Diefenbacher, Nikita Popov, Sophia M Blake, et al.
Nature Communications|October 4, 2013
Miz1 is required to maintain autophagic fluxElmar Wolf, Anneli Gebhardt, Daisuke Kawauchi, et al.
Cell Cycle (Georgetown, Tex.)|December 4, 2010
TGFβ-dependent gene expression shows that senescence correlates with abortive differentiation along several lineages in Myc-induced lymphomasJudith Müller, Birgit Samans, Jan van Riggelen, et al.
Molecular Cell|February 19, 2011
The MK5/PRAK kinase and Myc form a negative feedback loop that is disrupted during colorectal tumorigenesisTheresia R Kress, Ian G Cannell, Arjan B Brenkman, et al.
Plos One|October 8, 2013
CIP2A influences survival in colon cancer and is critical for maintaining Myc expressionArmin Wiegering, Christina Pfann, Friedrich Wilhelm Uthe, et al.
Nucleic Acids Research|December 19, 2024
Phenotypic screens identify SCAF1 as critical activator of RNAPII elongation and global transcriptionPranjali Bhandare, Ashwin Narain, Julia Hofstetter, et al.
The Journal of Biological Chemistry|June 3, 2010
Sequential activation of NFAT and c-Myc transcription factors mediates the TGF-beta switch from a suppressor to a promoter of cancer cell proliferationGarima Singh, Shiv K Singh, Alexander König, et al.
Pageof 17