Showing results (1-10 of 10) with videos related to

Sort By:
Pageof 1
Human Molecular Genetics|January 2, 2017
Cytoplasmic poly-GA aggregates impair nuclear import of TDP-43 in C9orf72 ALS/FTLDBahram Khosravi, Hannelore Hartmann, Stephanie May, et al.
Acta Neuropathologica|August 21, 2015
Erratum to: Distribution of dipeptide repeat proteins in cellular models and C9orf72 mutation cases suggests link to transcriptional silencingMartin H Schludi, Stephanie May, Friedrich A Grässer, et al.
Acta Neuropathologica|June 19, 2015
Distribution of dipeptide repeat proteins in cellular models and C9orf72 mutation cases suggests link to transcriptional silencingMartin H Schludi, Stephanie May, Friedrich A Grässer, et al.
EMBO Molecular Medicine|May 24, 2018
A novel CHCHD10 mutation implicates a Mia40-dependent mitochondrial import deficit in ALSCarina Lehmer, Martin H Schludi, Linnea Ransom, et al.
Acta Neuropathologica|August 15, 2014
C9orf72 FTLD/ALS-associated Gly-Ala dipeptide repeat proteins cause neuronal toxicity and Unc119 sequestrationStephanie May, Daniel Hornburg, Martin H Schludi, et al.
Acta Neuropathologica|October 18, 2013
Bidirectional transcripts of the expanded C9orf72 hexanucleotide repeat are translated into aggregating dipeptide repeat proteinsKohji Mori, Thomas Arzberger, Friedrich A Grässer, et al.
EMBO Molecular Medicine|March 30, 2017
Antibodies inhibit transmission and aggregation of C9orf72 poly-GA dipeptide repeat proteinsQihui Zhou, Carina Lehmer, Meike Michaelsen, et al.
The EMBO Journal|September 14, 2016
TDP-43 loss of function inhibits endosomal trafficking and alters trophic signaling in neuronsBenjamin M Schwenk, Hannelore Hartmann, Alperen Serdaroglu, et al.
EMBO Molecular Medicine|April 15, 2017
Poly-GP in cerebrospinal fluid links C9orf72-associated dipeptide repeat expression to the asymptomatic phase of ALS/FTDCarina Lehmer, Patrick Oeckl, Jochen H Weishaupt, et al.
Acta Neuropathologica|April 15, 2017
Spinal poly-GA inclusions in a C9orf72 mouse model trigger motor deficits and inflammation without neuron lossMartin H Schludi, Lore Becker, Lillian Garrett, et al.
Pageof 1