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The Journal of Surgical Research|April 7, 2012
Recovery from decompensated heart failure is associated with a distinct, phase-dependent gene expression profileNancy M Andersen, William E Stansfield, Ru-hang Tang, et al.
Cardiovascular Research|February 22, 2005
Adenosine produces nitric oxide and prevents mitochondrial oxidant damage in rat cardiomyocytesZhelong Xu, Sung-Sik Park, Robert A Mueller, et al.
Circulation Research|February 20, 2010
Sent to destroy: the ubiquitin proteasome system regulates cell signaling and protein quality control in cardiovascular development and diseaseMonte S Willis, W H Davin Townley-Tilson, Eunice Y Kang, et al.
The Journal of Biological Chemistry|May 24, 2014
Co-activation of nuclear factor-κB and myocardin/serum response factor conveys the hypertrophy signal of high insulin levels in cardiac myoblastsRosalinda Madonna, Yong-Jian Geng, Roberto Bolli, et al.
European Heart Journal. Case Reports|March 3, 2023
Bilateral deafness, diabetes, and different types of cardiomyopathy in family members with m.3243A > g mutation: a case reportFlorian Seiler, Philipp Ruile, Martin Moser, et al.
American Journal of Translational Research|December 19, 2013
Genetic myostatin decrease in the golden retriever muscular dystrophy model does not significantly affect the ubiquitin proteasome system despite enhancing the severity of diseaseSteven W Cotten, Joe N Kornegay, Daniel J Bogan, et al.
Arteriosclerosis, Thrombosis, and Vascular Biology|April 29, 2006
Comparative effects of paclitaxel and rapamycin on smooth muscle migration and survival: role of AKT-dependent signalingCam Patterson, Sabeen Mapera, Hui-Hua Li, et al.
The Journal of Clinical Investigation|October 30, 2007
Atrogin-1 inhibits Akt-dependent cardiac hypertrophy in mice via ubiquitin-dependent coactivation of Forkhead proteinsHui-Hua Li, Monte S Willis, Pamela Lockyer, et al.
Brain Research. Molecular Brain Research|March 30, 2004
Retrograde transport of the glucocorticoid receptor in neurites requires dynamic assembly of complexes with the protein chaperone hsp90 and is linked to the CHIP component of the machinery for proteasomal degradationMario D Galigniana, Jennifer M Harrell, Paul R Housley, et al.
Cell Biochemistry and Function|April 5, 2013
Carboxyl terminus of Hsp70-interacting protein (CHIP) is required to modulate cardiac hypertrophy and attenuate autophagy during exerciseMonte S Willis, Jin-Na Min, Shaobin Wang, et al.
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