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Arteriosclerosis, Thrombosis, and Vascular Biology|July 4, 2015
Senescent Vascular Smooth Muscle Cells Drive Inflammation Through an Interleukin-1α-Dependent Senescence-Associated Secretory PhenotypeSarah E Gardner, Melanie Humphry, Martin R Bennett, et al.
The Journal of Biological Chemistry|September 2, 2015
Interleukin-1α Activity in Necrotic Endothelial Cells Is Controlled by Caspase-1 Cleavage of Interleukin-1 Receptor-2: IMPLICATIONS FOR ALLOGRAFT REJECTIONLaura C Burzynski, Melanie Humphry, Martin R Bennett, et al.
Arteriosclerosis, Thrombosis, and Vascular Biology|September 3, 2011
Smooth muscle cell apoptosis promotes vessel remodeling and repair via activation of cell migration, proliferation, and collagen synthesisHaixiang Yu, Murray C H Clarke, Nichola Figg, et al.
Nature Reviews. Cardiology|June 28, 2019
Vascular smooth muscle cells in atherosclerosisGemma L Basatemur, Helle F Jørgensen, Murray C H Clarke, et al.
Cardiovascular Research|December 11, 2013
The CCR5 chemokine receptor mediates vasoconstriction and stimulates intimal hyperplasia in human vessels in vitroJanet J Maguire, Katie L Jones, Rhoda E Kuc, et al.
Nature Medicine|August 8, 2006
Apoptosis of vascular smooth muscle cells induces features of plaque vulnerability in atherosclerosisMurray C H Clarke, Nichola Figg, Janet J Maguire, et al.
European Journal of Immunology|May 25, 2020
Cell surface IL-1α trafficking is specifically inhibited by interferon-γ, and associates with the membrane via IL-1R2 and GPI anchorsJulie N E Chan, Melanie Humphry, Lauren Kitt, et al.
Circulation Research|May 24, 2008
Chronic apoptosis of vascular smooth muscle cells accelerates atherosclerosis and promotes calcification and medial degenerationMurray C H Clarke, Trevor D Littlewood, Nichola Figg, et al.
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