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Martin R Pollak

Showing results (41-50 of 167) with videos related to

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BMC Genetics|July 2, 2010
Improved IBD detection using incomplete haplotype informationGiulio Genovese, Gregory Leibon, Martin R Pollak, et al.
Tissue Engineering. Part B, Reviews|September 20, 2021
Bioengineering Strategies to Develop Podocyte Culture SystemsSarah Williams, Joseph L Charest, Martin R Pollak, et al.
Journal of the American Society of Nephrology : JASN|April 27, 2013
Inverted formin 2 regulates actin dynamics by antagonizing Rho/diaphanous-related formin signalingHua Sun, Johannes Schlondorff, Henry N Higgs, et al.
Biomaterials|September 7, 2018
Guided tissue organization and disease modeling in a kidney tubule arrayBalajikarthick Subramanian, Oguzhan Kaya, Martin R Pollak, et al.
Plos One|November 19, 2010
α-actinin-4 is essential for maintaining the spreading, motility and contractility of fibroblastsHanshuang Shao, James H-C Wang, Martin R Pollak, et al.
Ebiomedicine|June 19, 2015
Human Kidney Disease-causing INF2 Mutations Perturb Rho/Dia Signaling in the GlomerulusHua Sun, Khaldoun I Al-Romaih, Calum A MacRae, et al.
Biophysical Journal|August 12, 2008
Dynamic viscoelasticity of actin cross-linked with wild-type and disease-causing mutant alpha-actinin-4Sabine M Volkmer Ward, Astrid Weins, Martin R Pollak, et al.
Journal of the American Society of Nephrology : JASN|October 15, 2011
Population-based risk assessment of APOL1 on renal diseaseDavid J Friedman, Julia Kozlitina, Giulio Genovese, et al.
Plos One|December 16, 2016
Functional Validation of an Alpha-Actinin-4 Mutation as a Potential Cause of an Aggressive Presentation of Adolescent Focal Segmental Glomerulosclerosis: Implications for Genetic TestingDi Feng, Julia M Steinke, Ramaswamy Krishnan, et al.
The FEBS Journal|March 7, 2016
Structural characterization of the C-terminal coiled-coil domains of wild-type and kidney disease-associated mutants of apolipoprotein L1Alok K Sharma, David J Friedman, Martin R Pollak, et al.
Pageof 17

Showing results (41-50 of 167) with videos related to

Sort By:
Pageof 17
BMC Genetics|July 2, 2010
Improved IBD detection using incomplete haplotype informationGiulio Genovese, Gregory Leibon, Martin R Pollak, et al.
Tissue Engineering. Part B, Reviews|September 20, 2021
Bioengineering Strategies to Develop Podocyte Culture SystemsSarah Williams, Joseph L Charest, Martin R Pollak, et al.
Journal of the American Society of Nephrology : JASN|April 27, 2013
Inverted formin 2 regulates actin dynamics by antagonizing Rho/diaphanous-related formin signalingHua Sun, Johannes Schlondorff, Henry N Higgs, et al.
Biomaterials|September 7, 2018
Guided tissue organization and disease modeling in a kidney tubule arrayBalajikarthick Subramanian, Oguzhan Kaya, Martin R Pollak, et al.
Plos One|November 19, 2010
α-actinin-4 is essential for maintaining the spreading, motility and contractility of fibroblastsHanshuang Shao, James H-C Wang, Martin R Pollak, et al.
Ebiomedicine|June 19, 2015
Human Kidney Disease-causing INF2 Mutations Perturb Rho/Dia Signaling in the GlomerulusHua Sun, Khaldoun I Al-Romaih, Calum A MacRae, et al.
Biophysical Journal|August 12, 2008
Dynamic viscoelasticity of actin cross-linked with wild-type and disease-causing mutant alpha-actinin-4Sabine M Volkmer Ward, Astrid Weins, Martin R Pollak, et al.
Journal of the American Society of Nephrology : JASN|October 15, 2011
Population-based risk assessment of APOL1 on renal diseaseDavid J Friedman, Julia Kozlitina, Giulio Genovese, et al.
Plos One|December 16, 2016
Functional Validation of an Alpha-Actinin-4 Mutation as a Potential Cause of an Aggressive Presentation of Adolescent Focal Segmental Glomerulosclerosis: Implications for Genetic TestingDi Feng, Julia M Steinke, Ramaswamy Krishnan, et al.
The FEBS Journal|March 7, 2016
Structural characterization of the C-terminal coiled-coil domains of wild-type and kidney disease-associated mutants of apolipoprotein L1Alok K Sharma, David J Friedman, Martin R Pollak, et al.
Pageof 17