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Leukemia|May 18, 2022
Pediatric T-ALL type-1 and type-2 relapses develop along distinct pathways of clonal evolutionPaulina Richter-Pechańska, Joachim B Kunz, Tobias Rausch, et al.
Journal of Clinical Oncology : Official Journal of the American Society of Clinical Oncology|December 14, 2023
Four Additional Doses of PEG-L-Asparaginase During the Consolidation Phase in the AIEOP-BFM ALL 2009 Protocol Do Not Improve Outcome and Increase Toxicity in High-Risk ALL: Results of a Randomized StudyValentino Conter, Maria Grazia Valsecchi, Gunnar Cario, et al.
Proceedings of the National Academy of Sciences of the United States of America|May 3, 2017
Suppressors and activators of JAK-STAT signaling at diagnosis and relapse of acute lymphoblastic leukemia in Down syndromeOmer Schwartzman, Angela Maria Savino, Michael Gombert, et al.
Leukemia|March 8, 2019
PAX5 biallelic genomic alterations define a novel subgroup of B-cell precursor acute lymphoblastic leukemiaLorenz Bastian, Michael P Schroeder, Cornelia Eckert, et al.
Scientific Reports|October 15, 2015
The 9p21.3 risk of childhood acute lymphoblastic leukaemia is explained by a rare high-impact variant in CDKN2AJayaram Vijayakrishnan, Marc Henrion, Anthony V Moorman, et al.
Blood Advances|October 25, 2019
Pediatric ALL relapses after allo-SCT show high individuality, clonal dynamics, selective pressure, and druggable targetsJessica I Hoell, Sebastian Ginzel, Michaela Kuhlen, et al.
Nature Communications|November 27, 2019
Identification of four novel associations for B-cell acute lymphoblastic leukaemia riskJayaram Vijayakrishnan, Maoxiang Qian, James B Studd, et al.
Journal of Clinical Oncology : Official Journal of the American Society of Clinical Oncology|January 6, 2021
Clinical Implications of Minimal Residual Disease Detection in Infants With KMT2A-Rearranged Acute Lymphoblastic Leukemia Treated on the Interfant-06 ProtocolJanine Stutterheim, Inge M van der Sluis, Paola de Lorenzo, et al.
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