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Assay and Drug Development Technologies|December 8, 2010
Application of G protein-coupled receptor-heteromer identification technology to monitor β-arrestin recruitment to G protein-coupled receptor heteromersHeng B See, Ruth M Seeber, Martina Kocan, et al.
International Journal of Molecular Sciences|February 15, 2022
A Real-Time, Plate-Based BRET Assay for Detection of cGMP in Primary CellsAdam L Valkovic, Martina Kocan, Brad Hoare, et al.
Frontiers in Pharmacology|April 12, 2016
The Anti-fibrotic Actions of Relaxin Are Mediated Through a NO-sGC-cGMP-Dependent Pathway in Renal Myofibroblasts In Vitro and Enhanced by the NO Donor, Diethylamine NONOateChao Wang, Barbara K Kemp-Harper, Martina Kocan, et al.
Molecular Endocrinology (Baltimore, Md.)|January 31, 2009
Agonist-independent interactions between beta-arrestins and mutant vasopressin type II receptors associated with nephrogenic syndrome of inappropriate antidiuresisMartina Kocan, Heng B See, Natália G Sampaio, et al.
Pharmacology Research & Perspectives|September 29, 2018
Real-time examination of cAMP activity at relaxin family peptide receptors using a BRET-based biosensorAdam L Valkovic, Miranda B Leckey, Alice R Whitehead, et al.
Journal of Medicinal Chemistry|November 6, 2013
Minimum active structure of insulin-like peptide 5Alessia Belgi, Ross A D Bathgate, Martina Kocan, et al.
Pharmacology & Therapeutics|February 20, 2018
The relaxin receptor as a therapeutic target - perspectives from evolution and drug targetingRoss A D Bathgate, Martina Kocan, Daniel J Scott, et al.
Naunyn-Schmiedeberg'S Archives of Pharmacology|November 27, 2016
The actions of relaxin family peptides on signal transduction pathways activated by the relaxin family peptide receptor RXFP4Sheng Y Ang, Dana S Hutchinson, Bronwyn A Evans, et al.
Biochemical Pharmacology|April 12, 2021
GPR55 regulates the responsiveness to, but does not dimerise with, α1A-adrenoceptorsSarah K Walsh, Christopher Lipina, Sheng Y Ang, et al.
British Journal of Pharmacology|September 3, 2011
β(2)-Adrenoceptors increase translocation of GLUT4 via GPCR kinase sites in the receptor C-terminal tailNodi Dehvari, Dana S Hutchinson, Julia Nevzorova, et al.
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