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Mary E Hamby

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Neurotherapeutics : the Journal of the American Society for Experimental Neurotherapeutics|October 1, 2010
Reactive astrocytes as therapeutic targets for CNS disordersMary E Hamby, Michael V Sofroniew
Biochimica Et Biophysica Acta|August 5, 2008
Transcriptional regulation of neuronal differentiation: the epigenetic layer of complexityMary E Hamby, Volkan Coskun, Yi E Sun
Glia|July 8, 2010
Smad3-dependent signaling underlies the TGF-β1-mediated enhancement in astrocytic iNOS expressionMary E Hamby, James A Hewett, Sandra J Hewett
Glia|August 22, 2006
TGF-beta1 potentiates astrocytic nitric oxide production by expanding the population of astrocytes that express NOS-2Mary E Hamby, James A Hewett, Sandra J Hewett
Prostaglandins & Other Lipid Mediators|January 16, 2008
TGF-beta1 reduces the heterogeneity of astrocytic cyclooxygenase-2 and nitric oxide synthase-2 gene expression in a stimulus-independent mannerMary E Hamby, James A Hewett, Sandra J Hewett
Journal of Neuroscience Methods|August 18, 2005
Characterization of an improved procedure for the removal of microglia from confluent monolayers of primary astrocytesMary E Hamby, Tracy F Uliasz, Sandra J Hewett, et al.
Human Gene Therapy|March 28, 2002
Dopaminergic cell loss induced by human A30P alpha-synuclein gene transfer to the rat substantia nigraRonald L Klein, Michael A King, Mary E Hamby, et al.
Frontiers in Aging Neuroscience|February 27, 2019
The Role of APOE4 in Disrupting the Homeostatic Functions of Astrocytes and Microglia in Aging and Alzheimer's DiseaseCelia G Fernandez, Mary E Hamby, Morgan L McReynolds, et al.
Neurochemistry International|November 24, 2007
TGF beta 1 and TNF alpha potentiate nitric oxide production in astrocyte cultures by recruiting distinct subpopulations of cells to express NOS-2Mary E Hamby, Ariel R Gragnolati, Sandra J Hewett, et al.
Alzheimer'S & Dementia : the Journal of the Alzheimer'S Association|December 25, 2025
Drug DevelopmentJames E Galvin, Mary E Hamby, Jennifer Iaci, et al.
Pageof 4

Showing results (1-10 of 32) with videos related to

Sort By:
Pageof 4
Neurotherapeutics : the Journal of the American Society for Experimental Neurotherapeutics|October 1, 2010
Reactive astrocytes as therapeutic targets for CNS disordersMary E Hamby, Michael V Sofroniew
Biochimica Et Biophysica Acta|August 5, 2008
Transcriptional regulation of neuronal differentiation: the epigenetic layer of complexityMary E Hamby, Volkan Coskun, Yi E Sun
Glia|July 8, 2010
Smad3-dependent signaling underlies the TGF-β1-mediated enhancement in astrocytic iNOS expressionMary E Hamby, James A Hewett, Sandra J Hewett
Glia|August 22, 2006
TGF-beta1 potentiates astrocytic nitric oxide production by expanding the population of astrocytes that express NOS-2Mary E Hamby, James A Hewett, Sandra J Hewett
Prostaglandins & Other Lipid Mediators|January 16, 2008
TGF-beta1 reduces the heterogeneity of astrocytic cyclooxygenase-2 and nitric oxide synthase-2 gene expression in a stimulus-independent mannerMary E Hamby, James A Hewett, Sandra J Hewett
Journal of Neuroscience Methods|August 18, 2005
Characterization of an improved procedure for the removal of microglia from confluent monolayers of primary astrocytesMary E Hamby, Tracy F Uliasz, Sandra J Hewett, et al.
Human Gene Therapy|March 28, 2002
Dopaminergic cell loss induced by human A30P alpha-synuclein gene transfer to the rat substantia nigraRonald L Klein, Michael A King, Mary E Hamby, et al.
Frontiers in Aging Neuroscience|February 27, 2019
The Role of APOE4 in Disrupting the Homeostatic Functions of Astrocytes and Microglia in Aging and Alzheimer's DiseaseCelia G Fernandez, Mary E Hamby, Morgan L McReynolds, et al.
Neurochemistry International|November 24, 2007
TGF beta 1 and TNF alpha potentiate nitric oxide production in astrocyte cultures by recruiting distinct subpopulations of cells to express NOS-2Mary E Hamby, Ariel R Gragnolati, Sandra J Hewett, et al.
Alzheimer'S & Dementia : the Journal of the Alzheimer'S Association|December 25, 2025
Drug DevelopmentJames E Galvin, Mary E Hamby, Jennifer Iaci, et al.
Pageof 4