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Allergy|November 19, 2024
Endogenous Glucagon-Like Peptide-1 Receptor and Glucose-Dependent Insulinotropic Polypeptide Receptor Signaling Inhibits Aeroallergen-Induced Innate Airway InflammationShinji Toki, Masako Abney, Jian Zhang, et al.Journal of Immunology (Baltimore, Md. : 1950)|June 30, 2025
PGI2 signaling metabolically reprograms CD4 Th2 cells and represses allergic airway inflammationWeisong Zhou, Jian Zhang, Nowrin U Chowdhury, et al.Journal of Immunology (Baltimore, Md. : 1950)|May 7, 2025
PGI2 restricts trained ILC2 responses in allergic inflammationWeisong Zhou, Jian Zhang, Allison E Norlander, et al.JCI Insight|March 25, 2025
CFTR negatively reprograms Th2 cell responses, and CFTR potentiation restrains allergic airway inflammationMark Rusznak, Christopher M Thomas, Jian Zhang, et al.The Journal of Allergy and Clinical Immunology|August 14, 2025
Prostaglandin I<sub>2</sub> signaling restrains Treg cell ST2 expression by repressing β-catenin in allergic airway inflammationAllison E Norlander, Masako Abney, Jian Zhang, et al.Journal of Immunology (Baltimore, Md. : 1950)|September 24, 2025
Glucagon-like peptide-1 receptor signaling deficiency exacerbates hematopoietic stem cell graft rejection in miceMark Rusznak, Daniela Sierra-Hernandez, Catherine Dupuy, et al.Nature Communications|January 3, 2026
Genetic diversity of Collaborative Cross mice implicates FFAR3 as a target for ILC2 anti-inflammatory reprogrammingMark Rusznak, Shinji Toki, Yajing Hao, et al.Pageof 1