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Masao Endoh

Showing results (11-20 of 37) with videos related to

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British Journal of Pharmacology|February 20, 2014
Does levosimendan act as a Ca<sup>2+</sup> sensitizer or PDE3 inhibitor?: Commentary on Orstavik et al., Br J Pharmacol 171: 5169-5181Masao Endoh
Cardiovascular Research|February 19, 2008
Novel signalling cascade for cardiac hypertrophy activation by uncoupling and internalization of beta1-adrenoceptorsMasao Endoh
Circulation Research|August 20, 2016
Cardiac α1-Adrenoceptors and Inotropy: Myofilament Ca2+ Sensitivity, Intracellular Ca2+ Mobilization, Signaling Pathway, and Pathophysiological RelevanceMasao Endoh
Clinical Calcium|January 7, 2006
[Cav channels and Ca2+ antagonists: recent advances in molecular characteristics and pharmacology]Masao Endoh
Cardiovascular Drugs and Therapy|May 18, 2007
Basic and clinical characteristics of PDE 3 inhibitors as cardiotonic agentsMasao Endoh
Circulation Journal : Official Journal of the Japanese Circulation Society|November 5, 2008
Cardiac Ca2+ signaling and Ca2+ sensitizersMasao Endoh
Nihon Rinsho. Japanese Journal of Clinical Medicine|July 26, 2002
[Molecular mechanism of heart conduction system]Kuniaki Ishii, Masao Endoh
Expert Opinion on Pharmacotherapy|October 25, 2006
Acute heart failure: inotropic agents and their clinical usesMasao Endoh, Masatsugu Hori
Naunyn-Schmiedeberg'S Archives of Pharmacology|October 17, 2002
Effects of OR-1896, a metabolite of levosimendan, on force of contraction and Ca2+ transients under acidotic condition in aequorin-loaded canine ventricular myocardiumReiko Takahashi, Masao Endoh
British Journal of Pharmacology|June 14, 2005
Dual regulation of myofilament Ca2+ sensitivity by levosimendan in normal and acidotic conditions in aequorin-loaded canine ventricular myocardiumReiko Takahashi, Masao Endoh
Pageof 4

Showing results (11-20 of 37) with videos related to

Sort By:
Pageof 4
British Journal of Pharmacology|February 20, 2014
Does levosimendan act as a Ca<sup>2+</sup> sensitizer or PDE3 inhibitor?: Commentary on Orstavik et al., Br J Pharmacol 171: 5169-5181Masao Endoh
Cardiovascular Research|February 19, 2008
Novel signalling cascade for cardiac hypertrophy activation by uncoupling and internalization of beta1-adrenoceptorsMasao Endoh
Circulation Research|August 20, 2016
Cardiac α1-Adrenoceptors and Inotropy: Myofilament Ca2+ Sensitivity, Intracellular Ca2+ Mobilization, Signaling Pathway, and Pathophysiological RelevanceMasao Endoh
Clinical Calcium|January 7, 2006
[Cav channels and Ca2+ antagonists: recent advances in molecular characteristics and pharmacology]Masao Endoh
Cardiovascular Drugs and Therapy|May 18, 2007
Basic and clinical characteristics of PDE 3 inhibitors as cardiotonic agentsMasao Endoh
Circulation Journal : Official Journal of the Japanese Circulation Society|November 5, 2008
Cardiac Ca2+ signaling and Ca2+ sensitizersMasao Endoh
Nihon Rinsho. Japanese Journal of Clinical Medicine|July 26, 2002
[Molecular mechanism of heart conduction system]Kuniaki Ishii, Masao Endoh
Expert Opinion on Pharmacotherapy|October 25, 2006
Acute heart failure: inotropic agents and their clinical usesMasao Endoh, Masatsugu Hori
Naunyn-Schmiedeberg'S Archives of Pharmacology|October 17, 2002
Effects of OR-1896, a metabolite of levosimendan, on force of contraction and Ca2+ transients under acidotic condition in aequorin-loaded canine ventricular myocardiumReiko Takahashi, Masao Endoh
British Journal of Pharmacology|June 14, 2005
Dual regulation of myofilament Ca2+ sensitivity by levosimendan in normal and acidotic conditions in aequorin-loaded canine ventricular myocardiumReiko Takahashi, Masao Endoh
Pageof 4