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American Journal of Physiology. Renal Physiology|February 7, 2014
Downregulation of renal type IIa sodium-dependent phosphate cotransporter during lipopolysaccharide-induced acute inflammationShoko Ikeda, Hironori Yamamoto, Masashi Masuda, et al.
Journal of Lipid Research|July 20, 2024
All-trans retinoic acid induces lipophagy by reducing Rubicon in Hepa1c1c7 cellsAnh The Nguyen, Masashi Masuda, Yuki Mori, et al.
Journal of Clinical Biochemistry and Nutrition|October 12, 2020
Reduction of stearoyl-CoA desaturase (SCD) contributes muscle atrophy through the excess endoplasmic reticulum stress in chronic kidney diseaseYuki Niida, Masashi Masuda, Yuichiro Adachi, et al.
Life Science Alliance|March 23, 2022
All-trans retinoic acid changes muscle fiber type via increasing GADD34 dependent on MAPK signalYuichiro Adachi, Masashi Masuda, Iori Sakakibara, et al.
American Journal of Physiology. Renal Physiology|January 18, 2024
Elevated luminal inorganic phosphate suppresses intestinal Zn absorption in 5/6 nephrectomized ratsYosuke Okumura, Kotaro Abe, Shoko Sakai, et al.
The Journal of Nutritional Biochemistry|January 31, 2024
All-trans retinoic acid induces lipophagy through the activation of the AMPK-Beclin1 signaling pathway and reduces Rubicon expression in adipocytesYuki Mori, Masashi Masuda, Risa Yoshida-Shimizu, et al.
JCI Insight|November 5, 2016
Activating transcription factor-4 promotes mineralization in vascular smooth muscle cellsMasashi Masuda, Shinobu Miyazaki-Anzai, Audrey L Keenan, et al.
Journal of Clinical Biochemistry and Nutrition|July 29, 2024
Modulations of the mTORC2-GATA3 axis by an isorhamnetin activated endosomal-lysosomal system of the J774.1 macrophage-like cell lineMaiko Sakai, Kohta Ohnishi, Masashi Masuda, et al.
Journal of the American Heart Association|June 26, 2014
Endoplasmic reticulum stress effector CCAAT/enhancer-binding protein homologous protein (CHOP) regulates chronic kidney disease-induced vascular calcificationShinobu Miyazaki-Anzai, Masashi Masuda, Kimberly M Demos-Davies, et al.
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