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Biochemical and Biophysical Research Communications|July 9, 2016
The KYxxL motif in Rad17 protein is essential for the interaction with the 9-1-1 complexYasunori Fukumoto, Masayoshi Ikeuchi, Yuji Nakayama, et al.
FASEB Journal : Official Publication of the Federation of American Societies for Experimental Biology|December 28, 2020
The tumor suppressor LATS2 reduces v-Src-induced membrane blebs in a kinase activity-independent mannerMasayoshi Ikeuchi, Ryuzaburo Yuki, Youhei Saito, et al.
International Journal of Molecular Sciences|October 27, 2022
Rad17 Translocates to Nucleolus upon UV Irradiation through Nucleolar Localization Signal in the Central Basic DomainYasunori Fukumoto, Masayoshi Ikeuchi, Yuji Nakayama, et al.
International Journal of Molecular Sciences|April 19, 2017
Cytokinesis Failure Leading to Chromosome Instability in v-Src-Induced OncogenesisYuji Nakayama, Shuhei Soeda, Masayoshi Ikeuchi, et al.
International Journal of Molecular Sciences|February 9, 2020
Targeting Insulin-Like Growth Factor 1 Receptor Delays M-Phase Progression and Synergizes with Aurora B Inhibition to Suppress Cell ProliferationAkane Yamagishi, Yuki Ikeda, Masayoshi Ikeuchi, et al.
Cellular Signalling|November 23, 2016
v-Src-induced nuclear localization of YAP is involved in multipolar spindle formation in tetraploid cellsKeiko Kakae, Masayoshi Ikeuchi, Takahisa Kuga, et al.
International Journal of Molecular Sciences|June 9, 2016
v-Src Causes Chromosome Bridges in a Caffeine-Sensitive Manner by Generating DNA DamageMasayoshi Ikeuchi, Yasunori Fukumoto, Takuya Honda, et al.
The Journal of Biological Chemistry|June 26, 2021
Nuclear translocation promotes proteasomal degradation of human Rad17 protein through the N-terminal destruction boxesYasunori Fukumoto, Masayoshi Ikeuchi, Liang Qu, et al.
Experimental Cell Research|August 5, 2020
Kinase activity-independent role of EphA2 in the regulation of M-phase progressionYuichiro Kaibori, Kiriko Katayama, Yuka Tanaka, et al.
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