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Journal of Echocardiography|June 10, 2016
Long-term follow-up of a rare calcified cardiac tumor: a case reportNaotaka Okamoto, Naoki Mori, Yasuharu Lee, et al.Hypertension (Dallas, Tex. : 1979)|April 13, 2005
The antioxidant edaravone attenuates pressure overload-induced left ventricular hypertrophyIkuko Tsujimoto, Shungo Hikoso, Osamu Yamaguchi, et al.Eurointervention : Journal of Europcr in Collaboration with the Working Group on Interventional Cardiology of the European Society of Cardiology|March 8, 2014
Different serial changes in the neointimal condition of sirolimus-eluting stents and paclitaxel-eluting stents: an optical coherence tomographic studyDaisuke Nakamura, Yasuharu Lee, Takahiro Yoshimura, et al.Nature Medicine|April 24, 2007
The role of autophagy in cardiomyocytes in the basal state and in response to hemodynamic stressAtsuko Nakai, Osamu Yamaguchi, Toshihiro Takeda, et al.Biochemical and Biophysical Research Communications|January 5, 2005
CaMKII activates ASK1 and NF-kappaB to induce cardiomyocyte hypertrophyKazunori Kashiwase, Yoshiharu Higuchi, Shinichi Hirotani, et al.Circulation|January 16, 2008
Apoptosis signal-regulating kinase 1/p38 signaling pathway negatively regulates physiological hypertrophyMasayuki Taniike, Osamu Yamaguchi, Ikuko Tsujimoto, et al.Journal of Molecular and Cellular Cardiology|May 26, 2004
Ca(2+)-sensitive tyrosine kinase Pyk2/CAK beta-dependent signaling is essential for G-protein-coupled receptor agonist-induced hypertrophyShinichi Hirotani, Yoshiharu Higuchi, Kazuhiko Nishida, et al.Circulation|October 20, 2004
Pressure overload induces cardiac dysfunction and dilation in signal transducer and activator of transcription 6-deficient miceShungo Hikoso, Osamu Yamaguchi, Yoshiharu Higuchi, et al.Proceedings of the National Academy of Sciences of the United States of America|December 11, 2003
Targeted deletion of apoptosis signal-regulating kinase 1 attenuates left ventricular remodelingOsamu Yamaguchi, Yoshiharu Higuchi, Shinichi Hirotani, et al.Journal of the American College of Cardiology|August 23, 2008
Reduction in hemoglobin-oxygen affinity results in the improvement of exercise capacity in mice with chronic heart failureTetsuya Watanabe, Toshihiro Takeda, Shigemiki Omiya, et al.Pageof 4