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Epilepsia|April 6, 2011
Dravet syndrome: insights from in vitro experimental modelsMassimo MantegazzaScientific Reports|September 11, 2019
NaV1.2 haploinsufficiency in Scn2a knock-out mice causes an autistic-like phenotype attenuated with ageIsabelle Léna, Massimo MantegazzaNeuroscience Letters|November 14, 2017
Pathophysiological mechanisms of migraine and epilepsy: Similarities and differencesMassimo Mantegazza, Sandrine CestèleExpert Review of Clinical Pharmacology|November 25, 2011
Na+ channelopathies and epilepsy: recent advances and new perspectivesCarla Marini, Massimo MantegazzaThe Journal of Physiology|July 16, 2005
Beta-scorpion toxin effects suggest electrostatic interactions in domain II of voltage-dependent sodium channelsMassimo Mantegazza, Sandrine CestèleEpilepsia|January 7, 2020
SCN1A/NaV 1.1 channelopathies: Mechanisms in expression systems, animal models, and human iPSC modelsMassimo Mantegazza, Vania BroccoliNeurotherapeutics : the Journal of the American Society for Experimental Neurotherapeutics|March 26, 2014
Genetic epilepsy syndromes without structural brain abnormalities: clinical features and experimental modelsRenzo Guerrini, Carla Marini, Massimo MantegazzaEpilepsia|June 16, 2007
Epileptogenic channelopathies: experimental models of human pathologiesGiuliano Avanzini, Silvana Franceschetti, Massimo MantegazzaPhysiological Reviews|March 26, 2021
Sodium channelopathies of skeletal muscle and brainMassimo Mantegazza, Sandrine Cestèle, William A CatterallPlos One|February 6, 2013
Phenytoin inhibits the persistent sodium current in neocortical neurons by modifying its inactivation propertiesElisa Colombo, Silvana Franceschetti, Giuliano Avanzini, et al.Pageof 8